Aryl-hydrocarbon receptor activity modulates prolactin expression in the pituitary

Toxicology and Applied Pharmacology

Volumn 265, Issue 1, 2012, Pages 139-145

  Moran, Tyler B ^a   Brannick, Katherine E ^a   Raetzman, Lori T ^a  

^a UNIVERSITY OF ILLINOIS AT URBANA CHAMPAIGN   (United States)

Author keywords

AhR; Dioxin; Gonadotrope; Pituitary; Prolactin

Indexed keywords

AROMATIC HYDROCARBON RECEPTOR; BETA NAPHTHOFLAVONE; CYTOCHROME P450 1A1; DIOXIN; ENDOCRINE DISRUPTOR; MESSENGER RNA; PROLACTIN; TRANSFORMING GROWTH FACTOR BETA1;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CELL PROLIFERATION; CONTROLLED STUDY; GROWTH HORMONE SECRETING CELL; HYPOPHYSIS; KNOCKOUT MOUSE; MOUSE; NONHUMAN; PROLACTIN SECRETING CELL; PROTEIN EXPRESSION; TUMOR CELL LINE;

ANIMALS; BETA-NAPHTHOFLAVONE; CELL CYCLE; CELL LINE; CELL PROLIFERATION; CYTOCHROME P-450 CYP1A1; DIOXINS; ENDOCRINE DISRUPTORS; GROWTH HORMONE; MICE; MICE, KNOCKOUT; PITUITARY GLAND; POLYMERASE CHAIN REACTION; PROLACTIN; RATS; RECEPTORS, ARYL HYDROCARBON; RNA, MESSENGER; TRANSFORMING GROWTH FACTOR BETA1;

MUS; RATTUS;

EID: 84868301836     PISSN: 0041008X     EISSN: 10960333     Source Type: Journal    
DOI: 10.1016/j.taap.2012.08.026     Document Type: Article

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