Attenuation of oxidant damage in the postconditioned heart involves non-enzymatic response and partial catalytic protection

Experimental Physiology

Volumn 97, Issue 10, 2012, Pages 1119-1130

  Arreguín, Francisco ^a   García, Noemi ^a,b   Hernández Reséndiz, Sauri ^a   Buelna Chontal, Mabel ^a   Correa, Francisco ^a   Olín Sandoval, Viridiana ^a   Medina Campos, Omar Noel ^c   Pedraza Chaverrí, José ^c   Zazueta, Cecilia ^a  

^a INSTITUTO NACIONAL DE CARDIOLOGÍA IGNACIO CHÁVEZ   (Mexico)

^b ESCUELA DE MEDICINA Y CIENCIAS DE LA SALUD   (Mexico)

^c Universidad Nacional Autonoma de Mexico Facultad de Quimica   (Mexico)

Author keywords

[No Author keywords available]

Indexed keywords

CARBONYL DERIVATIVE; CATALASE; COPPER ZINC SUPEROXIDE DISMUTASE; DIETHYLTHIOCARBAMATE; GLUTATHIONE; GLUTATHIONE DISULFIDE; GLUTATHIONE PEROXIDASE; MANGANESE SUPEROXIDE DISMUTASE;

ANIMAL TISSUE; ANTIOXIDANT ACTIVITY; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVITY; HEART INFARCTION SIZE; HEART LEFT VENTRICLE PRESSURE; HEART MUSCLE NECROSIS; HEART MUSCLE REPERFUSION; HEART PROTECTION; HEART RATE; ISCHEMIC POSTCONDITIONING; ISOLATED HEART; LIPID PEROXIDATION; MALE; NONHUMAN; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; RAT; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; TISSUE HOMOGENATE;

EID: 84867010053     PISSN: 09580670     EISSN: 1469445X     Source Type: Journal    
DOI: 10.1113/expphysiol.2012.065763     Document Type: Article

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