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Dynamics of dna double-strand breaks revealed by clustering of damaged chromosome domains
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Functional targeting of dna damage to a nuclea r pore-associated sumo-dependent ubiquitin ligase
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53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility
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Mechanisms that regulate loca lization of a dna double-strand break to the nuclear periphery
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Doublestrand breaks in heterochromatin move outside of a dynamic hp 1a domain to complete recombinational repair
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Increased mobility of double-strand breaks requires mec1, rad9 and the homologous recombination machinery
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