Dissociation of structural and functional phenotypes in cardiac myosin-binding protein C conditional knockout mice

Circulation

Volumn 126, Issue 10, 2012, Pages 1194-1205

  Chen, Peter P ^a   Patel, Jitandrakumar R ^b   Powers, Patricia A ^a   Fitzsimons, Daniel P ^b   Moss, Richard L ^b  

^a UNIVERSITY OF WISCONSIN SCHOOL OF MEDICINE AND PUBLIC HEALTH   (United States)

^b UNIVERSITY OF WISCONSIN MADISON   (United States)

Author keywords

cardiomyopathy; heart failure; hypertrophy; myocardial contraction; remodeling

Indexed keywords

CARDIAC MYOSIN; CARDIAC MYOSIN BINDING PROTEIN C; CRE RECOMBINASE; ESTROGEN RECEPTOR; MYOSIN BINDING PROTEIN C; TAMOXIFEN; UNCLASSIFIED DRUG;

ALLELE; ANIMAL EXPERIMENT; AORTA CONSTRICTION; ARTICLE; CONTROLLED STUDY; DIASTOLE; FEMALE; GENE SILENCING; HEART DISEASE; HEART LEFT VENTRICLE DILATION; HEART MUSCLE CONTRACTILITY; HEART VENTRICLE HYPERTROPHY; MALE; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION;

AGE FACTORS; ANIMALS; ANTINEOPLASTIC AGENTS, HORMONAL; CARDIOMYOPATHY, HYPERTROPHIC; CARRIER PROTEINS; FEMALE; GENE EXPRESSION; HEART FAILURE; INTEGRASES; MALE; MICE; MICE, KNOCKOUT; MYOCARDIAL CONTRACTION; PHENOTYPE; TAMOXIFEN; VENTRICULAR FUNCTION, LEFT; VENTRICULAR PRESSURE; VENTRICULAR REMODELING;

EID: 84865861212     PISSN: 00097322     EISSN: 15244539     Source Type: Journal    
DOI: 10.1161/CIRCULATIONAHA.111.089219     Document Type: Article

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