Inhibition of B-NHEJ in plateau-phase cells is not a direct consequence of suppressed growth factor signaling

International Journal of Radiation Oncology Biology Physics

Volumn 84, Issue 2, 2012, Pages

  Singh, Satyendra K ^a   Bednar, Theresa ^a   Zhang, Lihua ^a   Wu, Wenqi ^b   Mladenov, Emil ^a   Iliakis, George ^a  

^a UNIVERSITY OF ESSEN MEDICAL SCHOOL   (Germany)

^b First Affiliated Hospital of Guangzhou Medical University   (China)

Author keywords

[No Author keywords available]

Indexed keywords

CATALYTIC SUBUNITS; CHEMICAL INHIBITION; CORE COMPONENTS; DNA-DEPENDENT PROTEIN KINASE; DOUBLE-STRAND BREAKS; EPIDERMAL GROWTH FACTOR RECEPTORS; ERROR PRONES; GROWTH ACTIVITY; GROWTH FACTOR; NONHOMOLOGOUS END JOINING; PROLIFERATING CELLS; PULSED FIELD GEL ELECTROPHORESIS; RADIATION RESPONSE; RAPID REDUCTION; REPAIR PATHWAYS; SERUM DEPRIVATION; TREATMENT DESIGN; UNDERLYING MECHANISM;

BODY FLUIDS; CELL PROLIFERATION; CYTOLOGY; DNA; ELECTROPHORESIS; ENZYMES; GROWTH (MATERIALS); REPAIR;

CELLS;

BACKUP NHEJ PROTEIN; CELL PROTEIN; EPIDERMAL GROWTH FACTOR RECEPTOR; UNCLASSIFIED DRUG;

ADAPTATION; ANIMAL CELL; ARTICLE; CANCER RADIOTHERAPY; CELL CYCLE PHASE; CELL PROLIFERATION; CLINICAL PROTOCOL; CONTROLLED STUDY; DNA REPAIR; DOUBLE STRANDED DNA BREAK; GEL ELECTROPHORESIS; MOUSE; NONHUMAN; PLATEAU PHASE; PRIORITY JOURNAL; PROTEIN FUNCTION; SIGNAL TRANSDUCTION;

ANIMALS; CELL GROWTH PROCESSES; CELL PROLIFERATION; CULTURE MEDIA, SERUM-FREE; DNA BREAKS, DOUBLE-STRANDED; DNA END-JOINING REPAIR; DNA-ACTIVATED PROTEIN KINASE; ELECTROPHORESIS, GEL, PULSED-FIELD; MICE; RECEPTOR, EPIDERMAL GROWTH FACTOR; TIME FACTORS;

EID: 84865647966     PISSN: 03603016     EISSN: 1879355X     Source Type: Journal    
DOI: 10.1016/j.ijrobp.2012.03.060     Document Type: Article

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