Reperfusion rather than ischemia drives the formation of ubiquitin aggregates after middle cerebral artery occlusion

Stroke

Volumn 43, Issue 8, 2012, Pages 2229-2235

  Hochrainer, Karin ^a   Jackman, Katherine ^a   Anrather, Josef ^a   Iadecola, Costantino ^a  

^a WEILL CORNELL MEDICAL COLLEGE   (United States)

Author keywords

middle cerebral artery occlusion; protein aggregation; reperfusion; stroke; ubiquitination

Indexed keywords

PROTEASOME; UBIQUITIN; UBIQUITINATED PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN INFARCTION SIZE; BRAIN ISCHEMIA; BRAIN LEVEL; CONTROLLED STUDY; CORPUS STRIATUM; DISEASE ASSOCIATION; DISEASE DURATION; MALE; MIDDLE CEREBRAL ARTERY OCCLUSION; MOUSE; NEOCORTEX; NONHUMAN; PRIORITY JOURNAL; PROTEIN AGGREGATION; PROTEIN CONTENT; REPERFUSION INJURY; UBIQUITINATION; WESTERN BLOTTING;

ANIMALS; BLOTTING, WESTERN; BRAIN; BRAIN ISCHEMIA; CORPUS STRIATUM; FLUOROMETRY; INFARCTION, MIDDLE CEREBRAL ARTERY; MALE; MICE; MICE, INBRED C57BL; NEOCORTEX; PEPTIDE HYDROLASES; PROTEASOME ENDOPEPTIDASE COMPLEX; REPERFUSION INJURY; SUBCELLULAR FRACTIONS; UBIQUITIN;

EID: 84864277301     PISSN: 00392499     EISSN: 15244628     Source Type: Journal    
DOI: 10.1161/STROKEAHA.112.650416     Document Type: Article

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