L-NAME cotreatment did prevent neither mitochondrial impairment nor behavioral abnormalities in adult Wistar rats treated with vitamin A supplementation

Fundamental and Clinical Pharmacology

Volumn 26, Issue 4, 2012, Pages 513-529

  de Oliveira, Marcos Roberto ^a   da Rocha, Ricardo Fagundes ^a   Schnorr, Carlos Eduardo ^a   Moreira, José Cláudio Fonseca ^a  

^a FEDERAL UNIVERSITY OF RIO GRANDE DO SUL   (Brazil)

Author keywords

Behavioral impairment; L NAME; Redox environment; Vitamin A

Indexed keywords

3 NITROTYROSINE; ADVANCED GLYCATION END PRODUCT; ADVANCED GLYCATION END PRODUCT RECEPTOR; ALPHA SYNUCLEIN; AMINE OXIDASE (FLAVIN CONTAINING); CYTOCHROME C OXIDASE; DOPAMINE 2 RECEPTOR; GLUTATHIONE TRANSFERASE; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); RETINOL PALMITATE; SUPEROXIDE; SUPEROXIDE DISMUTASE; UBIQUINOL CYTOCHROME C REDUCTASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BEHAVIOR DISORDER; CELL FUNCTION; CONTROLLED STUDY; CORPUS STRIATUM; DEGENERATIVE DISEASE; ENZYME ACTIVITY; FRONTAL CORTEX; HIPPOCAMPUS; MALE; MITOCHONDRION; NONHUMAN; OXIDATION REDUCTION POTENTIAL; PRIORITY JOURNAL; RAT; SUBSTANTIA NIGRA; TASK PERFORMANCE; VITAMIN SUPPLEMENTATION;

ALPHA-SYNUCLEIN; ANIMALS; ANXIETY DISORDERS; BEHAVIOR, ANIMAL; CENTRAL NERVOUS SYSTEM; DIETARY SUPPLEMENTS; ELECTRON TRANSPORT; GLUTATHIONE TRANSFERASE; HEAT-SHOCK PROTEINS; ILLNESS BEHAVIOR; LOCOMOTION; MALE; MANGANESE; MITOCHONDRIA; MONOAMINE OXIDASE; NEURODEGENERATIVE DISEASES; NG-NITROARGININE METHYL ESTER; NITRIC OXIDE; OXIDATION-REDUCTION; RATS; RATS, WISTAR; RECEPTORS, DOPAMINE; RECEPTORS, IMMUNOLOGIC; SUPEROXIDE DISMUTASE; SUPEROXIDES; TYROSINE; VITAMIN A;

EID: 84863558402     PISSN: 07673981     EISSN: 14728206     Source Type: Journal    
DOI: 10.1111/j.1472-8206.2011.00943.x     Document Type: Article

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