A critical role for MAP kinases in the control of Ah receptor complex activity

Toxicological Sciences

Volumn 82, Issue 1, 2004, Pages 80-87

  Tan, Zongqing ^a   Huang, Mingya ^a   Puga, Alvaro ^a   Xia, Ying ^a  

^a UNIVERSITY OF CINCINNATI MEDICAL CENTER   (United States)

Author keywords

2,3,7,8 Tetrachlorodibenzo p dioxin (TCDD); Ah receptor nuclear translocator (ARNT); Aromatic hydrocarbon receptor (AHR); Cytochrome p450 (CYP); Extracellular signal regulated kinase (ERK); Jun N terminal kinase (JNK)

Indexed keywords

2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; AROMATIC HYDROCARBON RECEPTOR; CYTOCHROME P450; CYTOCHROME P450 1A1; MITOGEN ACTIVATED PROTEIN KINASE;

AMINO TERMINAL SEQUENCE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DRUG METABOLISM; ENVIRONMENTAL EXPOSURE; GENE EXPRESSION REGULATION; GENETIC TRANSCRIPTION; LIVER; MOUSE; NONHUMAN; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; TESTIS; THYMUS; TISSUE DISTRIBUTION; WILD TYPE;

ANIMALS; ARYL HYDROCARBON RECEPTOR NUCLEAR TRANSLOCATOR; CELL LINE, TUMOR; CERCOPITHECUS AETHIOPS; CYTOCHROME P-450 CYP1A1; DNA-BINDING PROTEINS; ENVIRONMENTAL POLLUTANTS; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; FEMALE; GENE EXPRESSION REGULATION, ENZYMOLOGIC; LIVER; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MITOGEN-ACTIVATED PROTEIN KINASE 9; RECEPTORS, ARYL HYDROCARBON; TESTIS; TETRACHLORODIBENZODIOXIN; THYMUS GLAND; TRANSCRIPTION FACTORS;

EID: 8444245911     PISSN: 10966080     EISSN: None     Source Type: Journal    
DOI: 10.1093/toxsci/kfh228     Document Type: Article

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