Functional consequences of stably expressing a mutant calsequestrin (CASQ2 D307H) in the CASQ2 null background

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 302, Issue 1, 2012, Pages

  Kalyanasundaram, Anuradha ^a   Viatchenko Karpinski, Serge ^a   Belevych, Andriy E ^a   Lacombe, Veronique A ^b   Hwang, Hyun Seok ^c   Knollmann, Björn C ^c   Gyorke, Sandor ^a   Periasamy, Muthu ^a  

^a THE OHIO STATE UNIVERSITY COLLEGE OF MEDICINE   (United States)

^b OHIO STATE UNIVERSITY   (United States)

^c VANDERBILT UNIVERSITY   (United States)

Author keywords

Calcium release; Calcium transients; Catecholaminergic polymorphic ventricular tachycardia; Ryanodine receptor; Triadin 1

Indexed keywords

CALCIUM ION; CALSEQUESTRIN; ISOPRENALINE; MUTANT PROTEIN; PROTEIN; TRIADIN 1; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CALCIUM TRANSPORT; CONTROLLED STUDY; HEART MUSCLE CELL; MALE; MOLECULAR STABILITY; MOUSE; NONHUMAN; NULL ALLELE; NULL CELL; PHARMACOLOGICAL STIMULATION; POLYMORPHIC VENTRICULAR TACHYCARDIA; PRIORITY JOURNAL; SARCOPLASMIC RETICULUM; STABLE EXPRESSION; WILD TYPE;

ANIMALS; CALCIUM; CALCIUM SIGNALING; CALSEQUESTRIN; CARDIAC PACING, ARTIFICIAL; CARDIOTONIC AGENTS; CARRIER PROTEINS; DISEASE MODELS, ANIMAL; ELECTROCARDIOGRAPHY; GENOTYPE; ISOPROTERENOL; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; MUSCLE PROTEINS; MYOCYTES, CARDIAC; PHENOTYPE; POINT MUTATION; RYANODINE RECEPTOR CALCIUM RELEASE CHANNEL; SARCOPLASMIC RETICULUM; TACHYCARDIA, VENTRICULAR; TIME FACTORS;

EID: 84255167100     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00578.2011     Document Type: Article

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