Redox status and pro-survival/pro-apoptotic protein expression in the early cardiac hypertrophy induced by experimental hyperthyroidism

Cell Biochemistry and Function

Volumn 29, Issue 7, 2011, Pages 617-623

  Fernandes, R O ^a   Dreher, G J ^a   Schenkel, P C ^a   Fernandes, T R G ^a   Ribeiro, M F M ^a   Araujo, A S R ^a   Bello Klein A ^a  

^a FEDERAL UNIVERSITY OF RIO GRANDE DO SUL   (Brazil)

Author keywords

Akt; Bax Bcl 2; ERK1 2; Glutathione; Hydrogen peroxide; Oxidative stress

Indexed keywords

GLUTATHIONE; HYDROGEN PEROXIDE; LEVOTHYROXINE; MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN BAX; PROTEIN BCL 2; PROTEIN KINASE B; THIOREDOXIN; THIOREDOXIN REDUCTASE; TRANSCRIPTION FACTOR NRF2;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL SURVIVAL; CONTROLLED STUDY; ENZYME ACTIVITY; HEART VENTRICLE HYPERTROPHY; HYPERTHYROIDISM; LIVER CONGESTION; LUNG CONGESTION; MALE; NONHUMAN; OXIDATION REDUCTION STATE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; RAT; SIGNAL TRANSDUCTION; WESTERN BLOTTING;

ANIMALS; APOPTOSIS REGULATORY PROTEINS; BCL-2-ASSOCIATED X PROTEIN; BLOTTING, WESTERN; CARDIOMEGALY; CELL DEATH; DRINKING WATER; GLUTATHIONE; GLUTATHIONE DISULFIDE; HYDROGEN PEROXIDE; HYPERTHYROIDISM; MALE; MAP KINASE SIGNALING SYSTEM; MODELS, ANIMAL; OXIDATION-REDUCTION; PROTO-ONCOGENE PROTEINS C-AKT; RATS; RATS, WISTAR; THIOREDOXINS; THYROXINE;

RATTUS; RATTUS NORVEGICUS;

EID: 80053635384     PISSN: 02636484     EISSN: 10990844     Source Type: Journal    
DOI: 10.1002/cbf.1796     Document Type: Article

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