Redox regulation of myocardial Erk 1/2 phosphorylation in experimental hyperthyroidism: Role of thioredoxin-peroxiredoxin system

Journal of Cardiovascular Pharmacology

Volumn 56, Issue 5, 2010, Pages 513-517

  Da Rosa Araujo, Alex Sander ^a   Fernandes, Tania ^a   Ribeiro, Maria Flavia ^a   Khaper, Neelam ^b   Belló Klein, Adriane ^a  

^a FEDERAL UNIVERSITY OF RIO GRANDE DO SUL   (Brazil)

^b LAKEHEAD UNIVERSITY   (Canada)

Author keywords

ERK1 2; JNK; Nrf2; oxidative stress; thioredoxin; thyroxine

Indexed keywords

ALPHA TOCOPHEROL; GLUTATHIONE PEROXIDASE; GLUTATHIONE REDUCTASE; MITOGEN ACTIVATED PROTEIN KINASE 1; PEROXIREDOXIN 6; STRESS ACTIVATED PROTEIN KINASE; THIOREDOXIN 1; THYROXINE; TRANSCRIPTION FACTOR NRF2;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIOXIDANT ACTIVITY; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME PHOSPHORYLATION; HEART VENTRICLE HYPERTROPHY; HYPERTHYROIDISM; MALE; MODULATION; NONHUMAN; OXIDATION REDUCTION REACTION; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; REDOX STRESS;

ANIMALS; CARDIOMEGALY; GLUTATHIONE PEROXIDASE; GLUTATHIONE REDUCTASE; HYPERTHYROIDISM; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MALE; MAP KINASE SIGNALING SYSTEM; MITOGEN-ACTIVATED PROTEIN KINASE 1; MITOGEN-ACTIVATED PROTEIN KINASE 3; MYOCARDIUM; NF-E2-RELATED FACTOR 2; OXIDATION-REDUCTION; OXIDATIVE STRESS; PEROXIREDOXINS; PHOSPHORYLATION; RATS; RATS, WISTAR; THIOREDOXINS;

EID: 78650170566     PISSN: 01602446     EISSN: None     Source Type: Journal    
DOI: 10.1097/FJC.0b013e3181f50a70     Document Type: Article

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