Corin-deficient W-sh mice poorly tolerate increased cardiac afterload

Regulatory Peptides

Volumn 172, Issue 1-3, 2011, Pages 44-50

  Buckley, Cadie L ^a,b   Stokes, Alexander J ^a,b  

^a UNIVERSITY OF HAWAII   (United States)

^b UNIVERSITY OF HAWAII   (United States)

Author keywords

ANP; Atrial natriuretic peptide; Cardiac hypertrophy; Corin; Mast cell; W sh mice

Indexed keywords

ATRIAL NATRIURETIC FACTOR; CORIN; SERINE PROTEINASE; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL TISSUE; AORTA CONSTRICTION; ARTICLE; C57BL 6 MOUSE; CONTROLLED STUDY; DOPPLER ECHOCARDIOGRAPHY; HEART AFTERLOAD; HEART FAILURE; HEART HEMODYNAMICS; HEART LEFT VENTRICLE HYPERTROPHY; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN PROCESSING; TRANSTHORACIC ECHOCARDIOGRAPHY;

ANIMALS; AORTIC DISEASES; ATRIAL NATRIURETIC FACTOR; BLOTTING, WESTERN; CARDIOMEGALY; ECHOCARDIOGRAPHY, DOPPLER; MALE; MICE; MICE, INBRED C57BL; MICE, MUTANT STRAINS; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; SERINE ENDOPEPTIDASES; VENTRICULAR DYSFUNCTION, LEFT;

MUS;

EID: 80053568825     PISSN: 01670115     EISSN: 18731686     Source Type: Journal    
DOI: 10.1016/j.regpep.2011.08.006     Document Type: Article

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