Protein C mutation (A267T) results in ER retention and unfolded protein response activation

PLoS ONE

Volumn 6, Issue 8, 2011, Pages

  Tjeldhorn, Lena ^a,b   Iversen, Nina ^a   Sandvig, Kirsten ^a,b,c   Bergan, Jonas ^a,b,c   Sandset, Per Morten ^a,b   Skretting, Grethe ^a  

^a OSLO UNIVERSITY HOSPITAL   (Norway)

^b UNIVERSITY OF OSLO   (Norway)

^c UNIVERSITY OF OSLO   (Norway)

Author keywords

[No Author keywords available]

Indexed keywords

CALRETICULIN; CHAPERONE; DNA; GLUCOSE REGULATED PROTEIN 94; IMMUNOGLOBULIN BINDING FACTOR; INITIATION FACTOR 2ALPHA; PROTEASOME; PROTEIN C;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL LEVEL; CHO CELL; COMPLEX FORMATION; CONTROLLED STUDY; DNA FRAGMENTATION; ENDOPLASMIC RETICULUM; ENDOPLASMIC RETICULUM STRESS; FEMALE; GENE MUTATION; GENETIC TRANSFECTION; NONHUMAN; PATHOPHYSIOLOGY; PROTEIN C DEFICIENCY; PROTEIN DEGRADATION; PROTEIN PHOSPHORYLATION; PROTEIN SECRETION; PROTEIN VARIANT; SIGNAL TRANSDUCTION; STABLE EXPRESSION; UNFOLDED PROTEIN RESPONSE; WESTERN BLOTTING; WILD TYPE; ANIMAL; GENETICS; HAMSTER; HUMAN; METABOLISM; MUTATION; PHYSIOLOGY;

EUKARYOTA;

ANIMALS; APOPTOSIS; BLOTTING, WESTERN; CHO CELLS; CRICETINAE; ENDOPLASMIC RETICULUM; ENDOPLASMIC RETICULUM STRESS; HUMANS; MUTATION; PROTEIN C; UNFOLDED PROTEIN RESPONSE;

EID: 80052060531     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0024009     Document Type: Article

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