A transgenic mouse model for Alzheimer's disease has impaired synaptic gain but normal synaptic dynamics

Neuroscience Letters

Volumn 500, Issue 3, 2011, Pages 212-215

  Ricoy, Ulises M ^a   Mao, Peizhong ^b   Manczak, Maria ^b   Reddy, P Hemachandra ^b   Frerking, Matthew E ^a  

^a OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

^b OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

Author keywords

Alzheimer's; Amyloid; Glutamate; Hippocampus; Synaptic

Indexed keywords

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; EXCITATION; HIPPOCAMPUS; MOUSE; NEUROTRANSMISSION; NONHUMAN; PRIORITY JOURNAL; STIMULUS; SYNAPSE; SYNAPTIC TRANSMISSION; WILD TYPE;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; EXCITATORY POSTSYNAPTIC POTENTIALS; HIPPOCAMPUS; MICE; MICE, TRANSGENIC; PRESENILIN-1; SYNAPSES; SYNAPTIC TRANSMISSION;

EID: 79960629174     PISSN: 03043940     EISSN: 18727972     Source Type: Journal    
DOI: 10.1016/j.neulet.2011.06.043     Document Type: Article

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