Cross-bridge kinetics in myofibrils containing familial hypertrophic cardiomyopathy R58Q mutation in the regulatory light chain of myosin

Journal of Theoretical Biology

Volumn 284, Issue 1, 2011, Pages 71-81

  Mettikolla, P ^a   Calander, N ^a,b   Luchowski, R ^a   Gryczynski, I ^a   Gryczynski, Z ^a   Zhao, J ^c   Szczesna Cordary, D ^c   Borejdo, J ^a  

^a UNIVERSITY OF NORTH TEXAS HEALTH SCIENCE CENTER   (United States)

^b MACQUARIE UNIVERSITY   (Australia)

^c UNIVERSITY OF MIAMI MILLER SCHOOL OF MEDICINE   (United States)

Author keywords

Autocorrelation function; Familial hypertrophic cardiomyopathy; Muscle

Indexed keywords

ACTIN; ARGININE; GLUTAMINE;

BLOOD; CARDIOVASCULAR DISEASE; CROSS SECTION; MUSCLE; MUTATION; PROTEIN;

ACTIN MYOSIN INTERACTION; ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; FAMILIAL HYPERTROPHIC CARDIOMYOPATHY; GENE; GENE MUTATION; HEART CONTRACTION; HEART MUSCLE CELL; MALE; MOUSE; MUSCLE FIBRIL; MUSCLE FORCE; MUSCLE ISOMETRIC CONTRACTION; NONHUMAN; PRIORITY JOURNAL; RLC GENE; WILD TYPE;

ANIMALS; CARDIOMYOPATHY, HYPERTROPHIC, FAMILIAL; FEMALE; MALE; MICE; MICE, TRANSGENIC; MICROSCOPY, CONFOCAL; MYOCARDIAL CONTRACTION; MYOCARDIUM; MYOFIBRILS; MYOSIN LIGHT CHAINS; ORIENTATION; POINT MUTATION;

MUS;

EID: 79959966065     PISSN: 00225193     EISSN: 10958541     Source Type: Journal    
DOI: 10.1016/j.jtbi.2011.06.014     Document Type: Article

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