The N-terminal region of RECQL4 lacking the helicase domain is both essential and sufficient for the viability of vertebrate cells. Role of the N-terminal region of RECQL4 in cells

Biochimica et Biophysica Acta - Molecular Cell Research

Volumn 1813, Issue 3, 2011, Pages 473-479

  Abe, Takuya ^a   Yoshimura, Akari ^a,b   Hosono, Yoshifumi ^a   Tada, Shusuke ^a,c   Seki, Masayuki ^a   Enomoto, Takemi ^a,b  

^a TOHOKU UNIVERSITY   (Japan)

^b MUSASHINO UNIVERSITY   (Japan)

^c TEIKYO HEISEI UNIVERSITY   (Japan)

Author keywords

DNA repair; RecQ helicase; RecQL4; Rothmund Thomson syndrome; Sld2

Indexed keywords

DOXYCYCLINE; HELICASE; RECQ HELICASE; RECQL4 PROTEIN; UNCLASSIFIED DRUG;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL CULTURE; CELL DEATH; CELL FUNCTION; CELL GROWTH; CELL LINE; CELL VIABILITY; CHICKEN; DNA DAMAGE; DNA REPLICATION; DOUBLE STRANDED DNA BREAK; GENE EXPRESSION; GENE INACTIVATION; NONHUMAN; PRIORITY JOURNAL; SACCHAROMYCES CEREVISIAE;

ANIMALS; ANTI-BACTERIAL AGENTS; CELL DEATH; CELL LINE; CELL SURVIVAL; CHICKENS; DNA HELICASES; DNA REPAIR; DOXYCYCLINE; GENE EXPRESSION REGULATION; GENE KNOCKOUT TECHNIQUES; HUMANS; PROTEIN STRUCTURE, TERTIARY; RECQ HELICASES; ROTHMUND-THOMSON SYNDROME;

SACCHAROMYCES CEREVISIAE; VERTEBRATA;

EID: 79551685040     PISSN: 01674889     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.bbamcr.2011.01.001     Document Type: Article

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