Direct and indirect roles of RECQL4 in modulating base excision repair capacity

Human Molecular Genetics

Volumn 18, Issue 18, 2009, Pages 3470-3483

  Schurman, Shepherd H ^a   Hedayati, Mohammad ^a,d   Wang, Zheng Ming ^a   Singh, Dharmendra K ^a   Speina, Elzbieta ^a   Zhang, Yongqing ^b   Becker, Kevin ^b   Macris, Margaret ^c   Sung, Patrick ^c   Wilson III, David M ^a   Croteau, Deborah L ^a   Bohr, Vilhelm A ^a  

^a NATIONAL INSTITUTE ON AGING   (United States)

^b INSTITUTE OF BIOCHEMISTRY AND BIOPHYSICS   (Poland)

^c YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^d JOHNS HOPKINS UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DNA DIRECTED DNA POLYMERASE BETA; DNA FORMAMIDOPYRIMIDINE GLYCOSYLASE; FLAP ENDONUCLEASE; RECQ HELICASE; RECQL4 HELICASE; UNCLASSIFIED DRUG;

ARTICLE; CONTROLLED STUDY; DNA DAMAGE; DNA REPAIR; DNA STRAND BREAKAGE; ENZYME ACTIVITY; EXCISION REPAIR; FIBROBLAST; FRAMESHIFT MUTATION; GENE FREQUENCY; GENE LOCUS; GENE MUTATION; HUMAN; HUMAN CELL; MICROARRAY ANALYSIS; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN LOCALIZATION; PROTEIN PROTEIN INTERACTION; ROTHMUND THOMSON SYNDROME; UPREGULATION;

BIOCATALYSIS; CELLS, CULTURED; DNA DAMAGE; DNA POLYMERASE BETA; DNA REPAIR; DNA-(APURINIC OR APYRIMIDINIC SITE) LYASE; DNA-BINDING PROTEINS; GUANINE; HUMANS; HYDROGEN PEROXIDE; OLIGONUCLEOTIDE ARRAY SEQUENCE ANALYSIS; PYRIMIDINES; RECQ HELICASES; RNA, SMALL INTERFERING; ROTHMUND-THOMSON SYNDROME;

EID: 69449097526     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/ddp291     Document Type: Article

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