Chronic low-level Pb exposure during development decreases the expression of the voltage-dependent anion channel in auditory neurons of the brainstem

NeuroToxicology

Volumn 31, Issue 6, 2010, Pages 662-673

  Prins, John M ^a   Brooks, Diane M ^a   Thompson, Charles M ^a   Lurie, Diana I ^a  

^a UNIVERSITY OF MONTANA   (United States)

Author keywords

Brain type creatine kinase; Lead; Medial nucleus of the trapezoid body; Proteomics; Superior olivary complex; Voltage dependent anion channel

Indexed keywords

CREATINE KINASE; LEAD; VOLTAGE DEPENDENT ANION CHANNEL;

ACOUSTIC NERVE FIBER; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BRAIN STEM; CONTROLLED STUDY; DEVELOPMENT; DRUG EXPOSURE; FEMALE; GLYCOLYSIS; LEAD BLOOD LEVEL; MALE; MOUSE; NONHUMAN; OXIDATIVE PHOSPHORYLATION; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEOMICS; UPREGULATION;

ANIMALS; AUDITORY PATHWAYS; BRAIN STEM; GENE EXPRESSION REGULATION, DEVELOPMENTAL; LEAD; MICE; MICE, INBRED CBA; NEURONS; RANDOM ALLOCATION; RATS; VOLTAGE-DEPENDENT ANION CHANNELS;

ANIMALIA; MUS;

EID: 78049454599     PISSN: 0161813X     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuro.2010.08.004     Document Type: Article

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