Common genetic variants of the mitochondrial trafficking system and mitochondrial uncoupling proteins affect the development of two slowly developing demyelinating disorders, leukoaraiosis and multiple sclerosis

Current Medicinal Chemistry

Volumn 17, Issue 30, 2010, Pages 3583-3590

  Szolnoki, Z ^a  

^a Pandy County Hospital   (Hungary)

Author keywords

Genetic variants; Kinesin motor proteins; Leukoaraiosis; Multiple sclerosis; Ucps

Indexed keywords

KINESIN; MITOCHONDRIAL PROTEIN; UNCOUPLING PROTEIN;

ARTICLE; CELL ENERGY; CELL STRESS; COLD EXPOSURE; DIET RESTRICTION; ENERGY METABOLISM; GENE MUTATION; GENETIC VARIABILITY; GLIA CELL; HUMAN; HYPOXIA; LEUKOARAIOSIS; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; MULTIPLE SCLEROSIS; NONHUMAN; PROTEIN SYNTHESIS;

DEMYELINATING DISEASES; ENERGY METABOLISM; HUMANS; ION CHANNELS; KINESIN; LEUKOARAIOSIS; MEMBRANE POTENTIAL, MITOCHONDRIAL; MITOCHONDRIA; MITOCHONDRIAL PROTEINS; MULTIPLE SCLEROSIS; NEUROGLIA;

EID: 77957964796     PISSN: 09298673     EISSN: None     Source Type: Journal    
DOI: 10.2174/092986710792927868     Document Type: Article

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