MK-801 administration during neonatal ethanol withdrawal attenuates interpositus cell loss and juvenile eyeblink conditioning deficits

Alcohol

Volumn 44, Issue 4, 2010, Pages 359-369

  Young, Brandt W ^a,b   Sengelaub, Dale R ^b   Steinmetz, Joseph E ^c  

^a NORTHWESTERN UNIVERSITY   (United States)

^b INDIANA UNIVERSITY   (United States)

^c OHIO STATE UNIVERSITY   (United States)

Author keywords

Development; Ethanol; Eyeblink conditioning; MK 801

Indexed keywords

ALCOHOL; DIZOCILPINE; N METHYL DEXTRO ASPARTIC ACID RECEPTOR;

ALCOHOL BLOOD LEVEL; ALCOHOL INTOXICATION; ALCOHOL WITHDRAWAL; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BEHAVIOR DISORDER; CELL COUNT; CELL DEATH; CELL LOSS; CEREBELLUM; CONDITIONED REFLEX; CONTROLLED STUDY; DRUG MECHANISM; EXCITOTOXICITY; EYELID REFLEX; FEMALE; INTERPOSITUS NUCLEUS; LONG EVANS RAT; MALE; NERVE CELL; NEUROPROTECTION; NEUROTOXICITY; NEWBORN; NONHUMAN; PERINATAL PERIOD; RAT; TASK PERFORMANCE; TERATOGENICITY;

AGE FACTORS; ALCOHOL DRINKING; ANIMALS; ANIMALS, NEWBORN; CELL COUNT; CONDITIONING, EYELID; DIZOCILPINE MALEATE; ETHANOL; FEMALE; MALE; RATS; RATS, LONG-EVANS; SUBSTANCE WITHDRAWAL SYNDROME;

RATTUS; RODENTIA;

EID: 77955422034     PISSN: 07418329     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.alcohol.2009.12.002     Document Type: Article

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