-
1
-
-
45749132521
-
The E1784K mutation in SCN5A is associated with mixed clinical phenotype of type 3 long QT syndrome
-
Makita N, Behr E, Shimizu W et.al. The E1784K mutation in SCN5A is associated with mixed clinical phenotype of type 3 long QT syndrome. J Clin Invest. 2008;118(6):2219-2229
-
(2008)
J Clin Invest
, vol.118
, Issue.6
, pp. 2219-2229
-
-
Makita, N.1
Behr, E.2
Shimizu, W.3
-
2
-
-
33845708766
-
Overlap syndrome of cardiac sodium channel disease in mice carrying the equivalent mutation of human SCN5A-1795insD
-
Remme CA, Verkerk AO, Nuyens D et.al. Overlap syndrome of cardiac sodium channel disease in mice carrying the equivalent mutation of human SCN5A-1795insD. Circulation. 2006;114(24):2584-2594
-
(2006)
Circulation
, vol.114
, Issue.24
, pp. 2584-2594
-
-
Remme, C.A.1
Verkerk, A.O.2
Nuyens, D.3
-
3
-
-
0034730085
-
The elusive link between LQT3 and Brugada syndrome: The role of flecainide challenge
-
Priori SG, Napolitano C, Schwartz PJ.et.al. The elusive link between LQT3 and Brugada syndrome: the role of flecainide challenge. Circulation. 2000;102(9):945-947
-
(2000)
Circulation
, vol.102
, Issue.9
, pp. 945-947
-
-
Priori, S.G.1
Napolitano, C.2
Schwartz, P.J.3
-
5
-
-
33846034935
-
Simulation of Brugada syndrome using cellular and three-dimensional whole-heart modeling approaches
-
Xia L, Zhang Y, Zhang H et.al. Simulation of Brugada syndrome using cellular and three-dimensional whole-heart modeling approaches. Physiol Meas. 2006;27(11):1125-1142
-
(2006)
Physiol Meas
, vol.27
, Issue.11
, pp. 1125-1142
-
-
Xia, L.1
Zhang, Y.2
Zhang, H.3
-
6
-
-
0033594970
-
Congenital long-QT syndrome caused by a novel mutation in a conserved acidic domain of the cardiac Na+ channel
-
Wei J, Wang DW, Alings M et.al. Congenital long-QT syndrome caused by a novel mutation in a conserved acidic domain of the cardiac Na+ channel. Circulation. 1999 22;99(24):3165-3171
-
(1999)
Circulation
, vol.22-99
, Issue.24
, pp. 3165-3171
-
-
Wei, J.1
Wang, D.W.2
Alings, M.3
-
7
-
-
6044255042
-
In silico study of action potential and QT interval shortening due to loss of inactivation of the cardiac rapid delayed rectifier potassium current
-
Zhang H, Hancox JC. In silico study of action potential and QT interval shortening due to loss of inactivation of the cardiac rapid delayed rectifier potassium current. Biochem Biophys Res Commun. 2004;322(2):693-699
-
(2004)
Biochem Biophys Res Commun
, vol.322
, Issue.2
, pp. 693-699
-
-
Zhang, H.1
Hancox, J.C.2
-
8
-
-
47049098424
-
Brugada syndrome
-
Benito B, Brugada R, Brugada J, Brugada P. Brugada syndrome. Prog Cardiovasc Dis. 2008;51(1):1-22.
-
(2008)
Prog Cardiovasc Dis
, vol.51
, Issue.1
, pp. 1-22
-
-
Benito, B.1
Brugada, R.2
Brugada, J.3
Brugada, P.4
-
9
-
-
0032879716
-
Cellular basis for the Brugada syndrome and other mechanisms of arrhythmogenesis associated with ST-segment elevation
-
Yan GX, Antzelevitch C. Cellular basis for the Brugada syndrome and other mechanisms of arrhythmogenesis associated with ST-segment elevation. Circulation. 1999;100:1660-1666. (Pubitemid 29477277)
-
(1999)
Circulation
, vol.100
, Issue.15
, pp. 1660-1666
-
-
Yan, G.-X.1
Antzelevitch, C.2
-
10
-
-
39749084614
-
Cellular mechanism and arrhythmogenic potential of T-wave alternans in the Brugada syndrome
-
DOI 10.1111/j.1540-8167.2007.01025.x
-
Fish JM, Antzelevitch C. Cellular mechanism and arrhythmogenic potential of T-wave alternans in the Brugada syndrome. J Cardiovasc Electrophysiol. 2008;19:301-308. (Pubitemid 351294618)
-
(2008)
Journal of Cardiovascular Electrophysiology
, vol.19
, Issue.3
, pp. 301-308
-
-
Fish, J.M.1
Antzelevitch, C.2
-
11
-
-
33846885702
-
Cell model for efficient simulation of wave propagation in human ventricular tissue under normal and pathological conditions
-
Ten Tusscher KH, Panfilov AV. Cell model for efficient simulation of wave propagation in human ventricular tissue under normal and pathological conditions. Phys Med Biol. 2006;51(23):6141-6156
-
(2006)
Phys Med Biol
, vol.51
, Issue.23
, pp. 6141-6156
-
-
Ten Tusscher, K.H.1
Panfilov, A.V.2
|