In silico study of action potential and QT interval shortening due to loss of inactivation of the cardiac rapid delayed rectifier potassium current

Biochemical and Biophysical Research Communications

Volumn 322, Issue 2, 2004, Pages 693-699

  Zhang, Henggui ^a   Hancox, Jules C ^b  

^a UNIVERSITY OF MANCHESTER   (United Kingdom)

^b SCHOOL OF MEDICAL SCIENCES   (United Kingdom)

Author keywords

Dispersion of repolarisation; Human ether a go go related gene; I Kr; K + channel; Long QT syndrome; QT interval; Rapid delayed rectifier; Short QT syndrome

Indexed keywords

ACTION POTENTIAL; ARTICLE; COMPUTER MODEL; COMPUTER SIMULATION; ELECTRIC POTENTIAL; ELECTROCARDIOGRAM; ENDOCARDIUM; EPICARDIUM; HEART CONDUCTION; HEART MUSCLE CELL; HEART REPOLARIZATION; HEART VENTRICLE CONTRACTION; POTASSIUM CURRENT; PRIORITY JOURNAL; QT INTERVAL; HEART MUSCLE; HUMAN; KINETICS; METABOLISM; PATCH CLAMP; PHYSIOLOGY;

ERG1 POTASSIUM CHANNEL; KCNH2 PROTEIN, HUMAN; POTASSIUM; POTASSIUM CHANNEL; POTASSIUM CHANNEL HERG; VOLTAGE GATED POTASSIUM CHANNEL;

ACTION POTENTIALS; COMPUTER SIMULATION; ETHER-A-GO-GO POTASSIUM CHANNELS; HUMANS; KINETICS; MYOCARDIUM; PATCH-CLAMP TECHNIQUES; POTASSIUM; POTASSIUM CHANNELS; POTASSIUM CHANNELS, VOLTAGE-GATED;

EID: 6044255042     PISSN: 0006291X     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.bbrc.2004.07.176     Document Type: Article

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