Mechanisms of epigenetic silencing of the Rassf1a gene during estrogen-induced breast carcinogenesis in ACI rats

Carcinogenesis

Volumn 31, Issue 3, 2010, Pages 376-381

  Starlard Davenport, Athena ^a   Tryndyak, Volodymyr P ^a   James, Smitha R ^b   Karpf, Adam R ^b   Latendresse, John R ^a   Beland, Frederick A ^a   Pogribny, Igor P ^a  

^a NATIONAL CENTER FOR TOXICOLOGICAL RESEARCH   (United States)

^b ROSWELL PARK CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ESTRADIOL; HISTONE H3; RAS ASSOCIATION DOMAIN FAMILY PROTEIN 1A; HISTONE; HISTONE LYSINE METHYLTRANSFERASE; HISTONE METHYLTRANSFERASE; TUMOR PROTEIN; TUMOR SUPPRESSOR PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUGUST COPENHAGEN IRISH RAT; BREAST CARCINOGENESIS; CONTROLLED STUDY; CPG ISLAND; DNA METHYLATION; EPIGENETICS; FEMALE; GENE REPRESSION; GENE SILENCING; MOLECULAR MECHANICS; NONHUMAN; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN METHYLATION; RAT; ANIMAL; BIOSYNTHESIS; CELL TRANSFORMATION; CHEMICALLY INDUCED DISORDER; DRUG EFFECT; EXON; EXPERIMENTAL NEOPLASM; GENE EXPRESSION REGULATION; GENETICS; HYPERPLASIA; METABOLISM; PATHOLOGY; PRECANCER; RANDOMIZATION; TUMOR SUPPRESSOR GENE; UDDER;

ANIMALS; CELL TRANSFORMATION, NEOPLASTIC; CPG ISLANDS; DNA METHYLATION; ESTRADIOL; EXONS; FEMALE; GENE EXPRESSION REGULATION, NEOPLASTIC; GENE SILENCING; GENES, TUMOR SUPPRESSOR; HISTONE-LYSINE N-METHYLTRANSFERASE; HISTONES; HYPERPLASIA; MAMMARY GLANDS, ANIMAL; MAMMARY NEOPLASMS, EXPERIMENTAL; NEOPLASM PROTEINS; PRECANCEROUS CONDITIONS; PROMOTER REGIONS, GENETIC; RANDOM ALLOCATION; RATS; RATS, INBRED ACI; TUMOR SUPPRESSOR PROTEINS;

EID: 77950896567     PISSN: 01433334     EISSN: 14602180     Source Type: Journal    
DOI: 10.1093/carcin/bgp304     Document Type: Article

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