-
1
-
-
30744450770
-
The role of natriuretic peptides in cardioprotection
-
Nishikimi T, Maeda N, Matsuoka H (2006) The role of natriuretic peptides in cardioprotection. Cardiovasc Res 69:318-328.
-
(2006)
Cardiovasc Res
, vol.69
, pp. 318-328
-
-
Nishikimi, T.1
Maeda, N.2
Matsuoka, H.3
-
2
-
-
13144306072
-
Hypertension, cardiac hypertrophy, and sudden death in mice lacking natriuretic peptide receptor a
-
Oliver PM, et al. (1997) Hypertension, cardiac hypertrophy, and sudden death in mice lacking natriuretic peptide receptor A. Proc Natl Acad Sci USA 94:14730-14735.
-
(1997)
Proc Natl Acad Sci USA
, vol.94
, pp. 14730-14735
-
-
Oliver, P.M.1
-
3
-
-
0028973389
-
Salt-resistant hypertension in mice lacking the guanylyl cyclase-A receptor for atrial natriuretic peptide
-
Lopez MJ, et al. (1995) Salt-resistant hypertension in mice lacking the guanylyl cyclase-A receptor for atrial natriuretic peptide. Nature 378:65-68.
-
(1995)
Nature
, vol.378
, pp. 65-68
-
-
Lopez, M.J.1
-
4
-
-
0035063196
-
Pressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A-deficient mice
-
Knowles JW, et al. (2001) Pressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A-deficient mice. J Clin Invest 107:975-984.
-
(2001)
J Clin Invest
, vol.107
, pp. 975-984
-
-
Knowles, J.W.1
-
5
-
-
0035956914
-
A genetic model provides evidence that the receptor for atrial natriuretic peptide (guanylyl cyclase-A) inhibits cardiac ventricular myocyte hypertrophy
-
DOI 10.1073/pnas.051625598
-
Kishimoto I, Rossi K, Garbers DL (2001) A genetic model provides evidence that the receptor for atrial natriuretic peptide (guanylyl cyclase-A) inhibits cardiac ventricular myocyte hypertrophy. Proc Natl Acad Sci USA 98:2703-2706. (Pubitemid 32209546)
-
(2001)
Proceedings of the National Academy of Sciences of the United States of America
, vol.98
, Issue.5
, pp. 2703-2706
-
-
Kishimoto, I.1
Rossi, K.2
Garbers, D.L.3
-
6
-
-
0038814317
-
Pressure-independent cardiac hypertrophy in mice with cardiomyocyte-restricted inactivation of the atrial natriuretic peptide receptor guanylyl cyclase-A
-
DOI 10.1172/JCI200317061
-
Holtwick R, et al. (2003) Pressure-independent cardiac hypertrophy in mice with cardiomyocyte-restricted inactivation of the atrial natriuretic peptide receptor guanylyl cyclase-A. J Clin Invest 111:1399-1407. (Pubitemid 36554711)
-
(2003)
Journal of Clinical Investigation
, vol.111
, Issue.9
, pp. 1399-1407
-
-
Holtwick, R.1
Van Eickels, M.2
Skryabin, B.V.3
Baba, H.A.4
Bubikat, A.5
Begrow, F.6
Schneider, M.D.7
Garbers, D.L.8
Kuhn, M.9
-
7
-
-
0032519668
-
Nitric oxide, atrial natriuretic peptide, and cyclic GMP inhibit the growth-promoting effects of norepinephrine in cardiac myocytes and fibroblasts
-
Calderone A, Thaik CM, Takahashi N, Chang DL, Colucci WS (1998) Nitric oxide, atrial natriuretic peptide, and cyclic GMP inhibit the growth-promoting effects of norepinephrine in cardiac myocytes and fibroblasts. J Clin Invest 101:812-818. (Pubitemid 28096078)
-
(1998)
Journal of Clinical Investigation
, vol.101
, Issue.4
, pp. 812-818
-
-
Calderone, A.1
Thaik, C.M.2
Takahashi, N.3
Chang, D.L.F.4
Colucci, W.S.5
-
8
-
-
0033954454
-
Inhibitory regulation of hypertrophy by endogenous atrial natriuretic peptide in cultured cardiac myocytes
-
Horio T, et al. (2000) Inhibitory regulation of hypertrophy by endogenous atrial natriuretic peptide in cultured cardiac myocytes. Hypertension 35:19-24. (Pubitemid 30054231)
-
(2000)
Hypertension
, vol.35
, Issue.1 I
, pp. 19-24
-
-
Horio, T.1
Nishikimi, T.2
Yoshihara, F.3
Matsuo, H.4
Takishita, S.5
Kangawa, K.6
-
9
-
-
33847647511
-
Npr1-regulated gene pathways contributing to cardiac hypertrophy and fibrosis
-
Ellmers LJ, et al. (2007) Npr1-regulated gene pathways contributing to cardiac hypertrophy and fibrosis. J Mol Endocrinol 38:245-257.
-
(2007)
J Mol Endocrinol
, vol.38
, pp. 245-257
-
-
Ellmers, L.J.1
-
10
-
-
0028943463
-
Genetic decreases in atrial natriuretic peptide and salt-sensitive hypertension
-
John SW, et al. (1995) Genetic decreases in atrial natriuretic peptide and salt-sensitive hypertension. Science 267:679-681.
-
(1995)
Science
, vol.267
, pp. 679-681
-
-
John, S.W.1
-
11
-
-
7244255799
-
Atrial natriuretic peptide dose-dependently inhibits pressure overload-induced cardiac remodeling
-
DOI 10.1161/01.HYP.0000144801.09557.4c
-
Franco V, et al. (2004) Atrial natriuretic peptide dose-dependently inhibits pressure overload-induced cardiac remodeling. Hypertension 44:746-750. (Pubitemid 39435248)
-
(2004)
Hypertension
, vol.44
, Issue.5
, pp. 746-750
-
-
Franco, V.1
Chen, Y.-F.2
Oparil, S.3
Feng, J.A.4
Wang, D.5
Hage, F.6
Perry, G.7
-
12
-
-
1342324042
-
Volume overload results in exaggerated cardiac hypertrophy in the atrial natriuretic peptide knockout mouse
-
Mori T, et al. (2004) Volume overload results in exaggerated cardiac hypertrophy in the atrial natriuretic peptide knockout mouse. Cardiovasc Res 61:771-779.
-
(2004)
Cardiovasc Res
, vol.61
, pp. 771-779
-
-
Mori, T.1
-
13
-
-
33645217120
-
Cardiac hypertrophy in transgenic rats expressing a dominant-negative mutant of the natriuretic peptide receptor B
-
Langenickel TH, et al. (2006) Cardiac hypertrophy in transgenic rats expressing a dominant-negative mutant of the natriuretic peptide receptor B. Proc Natl Acad Sci USA 103:4735-4740.
-
(2006)
Proc Natl Acad Sci USA
, vol.103
, pp. 4735-4740
-
-
Langenickel, T.H.1
-
14
-
-
41149122223
-
Atrial natriuretic peptide inhibits transforming growth factor beta-induced Smad signaling and myofibroblast transformation in mouse cardiac fibroblasts
-
Li P, et al. (2008) Atrial natriuretic peptide inhibits transforming growth factor beta-induced Smad signaling and myofibroblast transformation in mouse cardiac fibroblasts. Circ Res 102:185-192.
-
(2008)
Circ Res
, vol.102
, pp. 185-192
-
-
Li, P.1
-
15
-
-
0033695554
-
Effect of neutral endopeptidase inhibitor on endogenous atrial natriuretic peptide as a paracrine factor in cultured cardiac fibroblasts
-
Maki T, et al. (2000) Effect of neutral endopeptidase inhibitor on endogenous atrial natriuretic peptide as a paracrine factor in cultured cardiac fibroblasts. Br J Pharmacol 131:1204-1210.
-
(2000)
Br J Pharmacol
, vol.131
, pp. 1204-1210
-
-
Maki, T.1
-
18
-
-
0037059550
-
CGMP-dependent protein kinase I mediates the negative inotropic effect of cGMP in the murine myocardium
-
DOI 10.1161/hh0102.103222
-
Wegener JW, et al. (2002) cGMP-dependent protein kinase I mediates the negative inotropic effect of cGMP in the murine myocardium. Circ Res 90:18-20. (Pubitemid 34074603)
-
(2002)
Circulation Research
, vol.90
, Issue.1
, pp. 18-20
-
-
Wegener, J.W.1
Nawrath, H.2
Wolfsgruber, W.3
Kuhbandner, S.4
Werner, C.5
Hofmann, F.6
Feil, R.7
-
19
-
-
33845934673
-
CGMP-dependent protein kinase type I inhibits TAB1-p38 mitogen-activated protein kinase apoptosis signaling in cardiac myocytes
-
DOI 10.1074/jbc.M603416200
-
Fiedler B, et al. (2006) cGMP-dependent protein kinase type I inhibits TAB1-p38 mitogen-activated protein kinase apoptosis signaling in cardiac myocytes. J Biol Chem 281:32831-32840. (Pubitemid 46036838)
-
(2006)
Journal of Biological Chemistry
, vol.281
, Issue.43
, pp. 32831-32840
-
-
Fiedler, B.1
Feil, R.2
Hofmann, F.3
Willenbockel, C.4
Drexler, H.5
Smolenski, A.6
Lohmann, S.M.7
Wollert, K.C.8
-
20
-
-
0037143710
-
Inhibition of calcineurin-NFAT hypertrophy signaling by cGMP-dependent protein kinase type I in cardiac myocytes
-
Fiedler B, et al. (2002) Inhibition of calcineurin-NFAT hypertrophy signaling by cGMP-dependent protein kinase type I in cardiac myocytes. Proc Natl Acad Sci USA 99: 11363-11368.
-
(2002)
Proc Natl Acad Sci USA
, vol.99
, pp. 11363-11368
-
-
Fiedler, B.1
-
21
-
-
24044492874
-
Protein kinase G transmits the cardioprotective signal from cytosol to mitochondria
-
Costa AD, et al. (2005) Protein kinase G transmits the cardioprotective signal from cytosol to mitochondria. Circ Res 97:329-336.
-
(2005)
Circ Res
, vol.97
, pp. 329-336
-
-
Costa, A.D.1
-
22
-
-
0024453191
-
Decreased cardiac concentration of cGMP kinase in hypertensive animals. An index for cardiac vascularization?
-
Ecker T, et al. (1989) Decreased cardiac concentration of cGMP kinase in hypertensive animals. An index for cardiac vascularization? Circ Res 65:1361-1369.
-
(1989)
Circ Res
, vol.65
, pp. 1361-1369
-
-
Ecker, T.1
-
23
-
-
36349033482
-
Phosphodiesterase type 5: Expanding roles in cardiovascular regulation
-
Kass DA, Champion HC, Beavo JA (2007) Phosphodiesterase type 5: Expanding roles in cardiovascular regulation. Circ Res 101:1084-1095.
-
(2007)
Circ Res
, vol.101
, pp. 1084-1095
-
-
Kass, D.A.1
Champion, H.C.2
Beavo, J.A.3
-
24
-
-
34447265905
-
Biochemistry and physiology of cyclic nucleotide phosphodiesterases: Essential components in cyclic nucleotide signaling
-
Conti M, Beavo J (2007) Biochemistry and physiology of cyclic nucleotide phosphodiesterases: Essential components in cyclic nucleotide signaling. Annu Rev Biochem 76:481-511.
-
(2007)
Annu Rev Biochem
, vol.76
, pp. 481-511
-
-
Conti, M.1
Beavo, J.2
-
25
-
-
33750813323
-
Relation between contractile function and regulatory cardiac proteins in hypertrophied hearts
-
Stein B, et al. (1996) Relation between contractile function and regulatory cardiac proteins in hypertrophied hearts. Am J Physiol 270:H2021-H2028. (Pubitemid 126833607)
-
(1996)
American Journal of Physiology - Heart and Circulatory Physiology
, vol.39
, Issue.6
-
-
Stein, B.1
Bartel, S.2
Kirchhefer, U.3
Kokott, S.4
Krause, E.-G.5
Neumann, J.6
Schmitz, W.7
Scholz, H.8
-
26
-
-
0034617175
-
Cardiac intracellular calcium release channels: Role in heart failure
-
Marks AR (2000) Cardiac intracellular calcium release channels: Role in heart failure. Circ Res 87:8-11. (Pubitemid 30456928)
-
(2000)
Circulation Research
, vol.87
, Issue.1
, pp. 8-11
-
-
Marks, A.R.1
-
27
-
-
0037351716
-
Inhibition of cyclic GMP hydrolysis with zaprinast reduces basal and cyclic AMP-elevated L-type calcium current in guinea-pig ventricular myocytes
-
Ziolo MT, Lewandowski SJ, Smith JM, Romano FD, Wahler GM (2003) Inhibition of cyclic GMP hydrolysis with zaprinast reduces basal and cyclic AMP-elevated L-type calcium current in guinea-pig ventricular myocytes. Br J Pharmacol 138:986-994.
-
(2003)
Br J Pharmacol
, vol.138
, pp. 986-994
-
-
Ziolo, M.T.1
Lewandowski, S.J.2
Smith, J.M.3
Romano, F.D.4
Wahler, G.M.5
-
28
-
-
0026056572
-
Potentiation by cyclic GMP of beta-adrenergic effect on Ca2+ current in guinea-pig ventricular cells
-
Lond
-
Ono K, Trautwein W (1991) Potentiation by cyclic GMP of beta-adrenergic effect on Ca2+ current in guinea-pig ventricular cells. J Physiol (Lond) 443:387-404.
-
(1991)
J Physiol
, vol.443
, pp. 387-404
-
-
Ono, K.1
Trautwein, W.2
-
29
-
-
0030727850
-
Catecholamine-induced cardiac hypertrophy: Significance of proto-oncogene expression
-
Zimmer HG (1997) Catecholamine-induced cardiac hypertrophy: Significance of protooncogene expression. J Mol Med 75:849-859. (Pubitemid 27511561)
-
(1997)
Journal of Molecular Medicine
, vol.75
, Issue.11-12
, pp. 849-859
-
-
Zimmer, H.-G.1
-
30
-
-
42549128214
-
Epac mediates beta-adrenergic receptor-induced cardiomyocyte hypertrophy
-
Métrich M, et al. (2008) Epac mediates beta-adrenergic receptor-induced cardiomyocyte hypertrophy. Circ Res 102:959-965.
-
(2008)
Circ Res
, vol.102
, pp. 959-965
-
-
Métrich, M.1
-
31
-
-
59049107401
-
Critical role of transcription factor cyclic AMP response element modulator in beta1-adrenoceptor-mediated cardiac dysfunction
-
Lewin G, et al. (2009) Critical role of transcription factor cyclic AMP response element modulator in beta1-adrenoceptor-mediated cardiac dysfunction. Circulation 119:79-88.
-
(2009)
Circulation
, vol.119
, pp. 79-88
-
-
Lewin, G.1
-
32
-
-
0029057806
-
Nitric oxide-dependent parasympathetic signaling is due to activation of constitutive endothelial (type III) nitric oxide synthase in cardiac myocytes
-
Balligand JL, et al. (1995) Nitric oxide-dependent parasympathetic signaling is due to activation of constitutive endothelial (type III) nitric oxide synthase in cardiac myocytes. J Biol Chem 270:14582-14586.
-
(1995)
J Biol Chem
, vol.270
, pp. 14582-14586
-
-
Balligand, J.L.1
-
33
-
-
19944428363
-
CGMP catabolism by phosphodiesterase 5A regulates cardiac adrenergic stimulation by NOS3-dependent mechanism
-
DOI 10.1161/01.RES.0000152262.22968.72
-
Takimoto E, et al. (2005) cGMP catabolism by phosphodiesterase 5A regulates cardiac adrenergic stimulation by NOS3-dependent mechanism. Circ Res 96:100-109. (Pubitemid 40095759)
-
(2005)
Circulation Research
, vol.96
, Issue.1
, pp. 100-109
-
-
Takimoto, E.1
Champion, H.C.2
Belardi, D.3
Moslehi, J.4
Mongillo, M.5
Mergia, E.6
Montrose, D.C.7
Isoda, T.8
Aufiero, K.9
Zaccolo, M.10
Dostmann, W.R.11
Smith, C.J.12
Kass, D.A.13
-
34
-
-
20044363270
-
Chronic inhibition of cyclic GMP phosphodiesterase 5A prevents and reverses cardiac hypertrophy
-
DOI 10.1038/nm1175
-
Takimoto E, et al. (2005) Chronic inhibition of cyclic GMP phosphodiesterase 5A prevents and reverses cardiac hypertrophy. Nat Med 11:214-222. (Pubitemid 40321358)
-
(2005)
Nature Medicine
, vol.11
, Issue.2
, pp. 214-222
-
-
Takimoto, E.1
Champion, H.C.2
Li, M.3
Belardi, D.4
Ren, S.5
Rodriguez, E.R.6
Bedja, D.7
Gabrielson, K.L.8
Wang, Y.9
Kass, D.A.10
-
35
-
-
58149104494
-
Sildenafil stops progressive chamber, cellular, and molecular remodeling and improves calcium handling and function in hearts with pre-existing advanced hypertrophy caused by pressure overload
-
Nagayama T, et al. (2009) Sildenafil stops progressive chamber, cellular, and molecular remodeling and improves calcium handling and function in hearts with pre-existing advanced hypertrophy caused by pressure overload. J Am Coll Cardiol 53: 207-215.
-
(2009)
J Am Coll Cardiol
, vol.53
, pp. 207-215
-
-
Nagayama, T.1
-
36
-
-
10744225434
-
2+ channel regulation by cGMP-dependent protein kinase type I in adult cardiomyocytes from PKG I transgenic mice
-
DOI 10.1016/S0008-6363(03)00546-7
-
Schröder F, et al. (2003) Single L-type Ca(2+) channel regulation by cGMP-dependent protein kinase type I in adult cardiomyocytes from PKG I transgenic mice. Cardiovasc Res 60:268-277. (Pubitemid 37412360)
-
(2003)
Cardiovascular Research
, vol.60
, Issue.2
, pp. 268-277
-
-
Schroder, F.1
Klein, G.2
Fiedler, B.3
Bastein, M.4
Schnasse, N.5
Hillmer, A.6
Ames, S.7
Gambaryan, S.8
Drexler, H.9
Walter, U.10
Lohmann, S.M.11
Wollert, K.C.12
-
37
-
-
47649122637
-
Reversal of cardiac hypertrophy and fibrosis from pressure overload by tetrahydrobiopterin: Efficacy of recoupling nitric oxide synthase as a therapeutic strategy
-
Moens AL, et al. (2008) Reversal of cardiac hypertrophy and fibrosis from pressure overload by tetrahydrobiopterin: Efficacy of recoupling nitric oxide synthase as a therapeutic strategy. Circulation 117:2626-2636.
-
(2008)
Circulation
, vol.117
, pp. 2626-2636
-
-
Moens, A.L.1
-
38
-
-
0036153139
-
Gene transfer of cGMP-dependent protein kinase I enhances the antihypertrophic effects of nitric oxide in cardiomyocytes
-
Wollert KC, et al. (2002) Gene transfer of cGMP-dependent protein kinase I enhances the antihypertrophic effects of nitric oxide in cardiomyocytes. Hypertension 39:87-92.
-
(2002)
Hypertension
, vol.39
, pp. 87-92
-
-
Wollert, K.C.1
-
39
-
-
0027495714
-
Nitricoxide regulates cardiac Ca2+ current. Involvement of cGMP-inhibited and cGMP-stimulated phosphodiesterases through guanylyl cyclase activation
-
Méry PF, Pavoine C, Belhassen L, Pecker F, Fischmeister R (1993) Nitricoxide regulates cardiac Ca2+ current. Involvement of cGMP-inhibited and cGMP-stimulated phosphodiesterases through guanylyl cyclase activation. J Biol Chem 268:26286-26295.
-
(1993)
J Biol Chem
, vol.268
, pp. 26286-26295
-
-
Méry, P.F.1
Pavoine, C.2
Belhassen, L.3
Pecker, F.4
Fischmeister, R.5
-
40
-
-
26844470536
-
+ exchanger NHE-1 contributes to cardiac remodeling in atrial natriuretic peptide receptor-deficient mice
-
DOI 10.1161/CIRCULATIONAHA.105.542209
-
Kilic A, et al. (2005) Enhanced activity of the myocardial Na+/H+ exchanger NHE-1 contributes to cardiac remodeling in atrial natriuretic peptide receptor-deficient mice. Circulation 112:2307-2317. (Pubitemid 41464574)
-
(2005)
Circulation
, vol.112
, Issue.15
, pp. 2307-2317
-
-
Kilic, A.1
Velic, A.2
De Windt, L.J.3
Fabritz, L.4
Voss, M.5
Mitko, D.6
Zwiener, M.7
Baba, H.A.8
Van Eickels, M.9
Schlatter, E.10
Kuhn, M.11
-
41
-
-
26444507242
-
Atrial natriuretic peptide promotes cardiomyocyte survival by cGMP-dependent nuclear accumulation of zyxin and Akt
-
DOI 10.1172/JCI24280
-
Kato T, et al. (2005) Atrial natriuretic peptide promotes cardiomyocyte survival by cGMP-dependent nuclear accumulation of zyxin and Akt. J Clin Invest 115:2716-2730. (Pubitemid 41434399)
-
(2005)
Journal of Clinical Investigation
, vol.115
, Issue.10
, pp. 2716-2730
-
-
Kato, T.1
Muraski, J.2
Chen, Y.3
Tsujita, Y.4
Wall, J.5
Glembotski, C.C.6
Schaefer, E.7
Beckerle, M.8
Sussman, M.A.9
-
42
-
-
59649084739
-
Protein kinase G modulates human myocardial passive stiffness by phosphorylation of the titin springs
-
Krüger M, et al. (2009) Protein kinase G modulates human myocardial passive stiffness by phosphorylation of the titin springs. Circ Res 104:87-94.
-
(2009)
Circ Res
, vol.104
, pp. 87-94
-
-
Krüger, M.1
-
43
-
-
36348957856
-
Rescue of cGMP kinase I knockout mice by smooth muscle-specific expression of either isozyme
-
DOI 10.1161/CIRCRESAHA.107.154351
-
Weber S, et al. (2007) Rescue of cGMP kinase I knockout mice by smooth muscle specific expression of either isozyme. Circ Res 101:1096-1103. (Pubitemid 350146435)
-
(2007)
Circulation Research
, vol.101
, Issue.11
, pp. 1096-1103
-
-
Weber, S.1
Bernhard, D.2
Lukowski, R.3
Weinmeister, P.4
Worner, R.5
Wegener, J.W.6
Valtcheva, N.7
Feil, S.8
Schlossmann, J.9
Hofmann, F.10
Feil, R.11
-
44
-
-
35348870623
-
Cyclic GMP and protein kinase-G in myocardial ischaemia-reperfusion: Opportunities and obstacles for survival signaling
-
DOI 10.1038/sj.bjp.0707409, PII 0707409
-
Burley DS, Ferdinandy P, Baxter GF (2007) Cyclic GMP and protein kinase-G in myocardial ischaemia-reperfusion: Opportunities and obstacles for survival signaling. Br J Pharmacol 152:855-869. (Pubitemid 350100488)
-
(2007)
British Journal of Pharmacology
, vol.152
, Issue.6
, pp. 855-869
-
-
Burley, D.S.1
Ferdinandy, P.2
Baxter, G.F.3
-
45
-
-
57149142506
-
Cardioprotection: Nitric oxide, protein kinases, and mitochondria
-
Heusch G, Boengler K, Schulz R (2008) Cardioprotection: Nitric oxide, protein kinases, and mitochondria. Circulation 118:1915-1919.
-
(2008)
Circulation
, vol.118
, pp. 1915-1919
-
-
Heusch, G.1
Boengler, K.2
Schulz, R.3
-
46
-
-
0032101741
-
Defective smooth muscle regulation in cGMP kinase I-deficient mice
-
DOI 10.1093/emboj/17.11.3045
-
Pfeifer A, et al. (1998) Defective smooth muscle regulation in cGMP kinase I-deficient mice. EMBO J 17:3045-3051. (Pubitemid 28254377)
-
(1998)
EMBO Journal
, vol.17
, Issue.11
, pp. 3045-3051
-
-
Pfeifer, A.1
Klatt, P.2
Massberg, S.3
Ny, L.4
Sausbier, M.5
Hirneiss, C.6
Wang, G.-X.7
Korth, M.8
Aszodi, A.9
Andersson, K.-E.10
Krombach, F.11
Mayerhofer, A.12
Ruth, P.13
Fassler, R.14
Hofmann, F.15
-
47
-
-
4544320797
-
Distribution of IRAG and cGKI-isoforms in murine tissues
-
DOI 10.1016/j.febslet.2004.08.030, PII S0014579304010294
-
Geiselhöringer A, Gaisa M, Hofmann F, Schlossmann J (2004) Distribution of IRAG and cGKI-isoforms in murine tissues. FEBS Lett 575:19-22. (Pubitemid 39237675)
-
(2004)
FEBS Letters
, vol.575
, Issue.1-3
, pp. 19-22
-
-
Geiselhoringer, A.1
Gaisa, M.2
Hofmann, F.3
Schlossmann, J.4
-
48
-
-
0028303913
-
CAMP- And cGMP-dependent protein kinase phosphorylation sites of the focal adhesion vasodilator-stimulated phosphoprotein (VASP) in vitro and in intact human platelets
-
Butt E, et al. (1994) cAMP- and cGMP-dependent protein kinase phosphorylation sites of the focal adhesion vasodilator-stimulated phosphoprotein (VASP) in vitro and in intact human platelets. J Biol Chem 269:14509-14517. (Pubitemid 24194005)
-
(1994)
Journal of Biological Chemistry
, vol.269
, Issue.20
, pp. 14509-14517
-
-
Butt, E.1
Abel, K.2
Krieger, M.3
Palm, D.4
Hoppe, V.5
Hoppe, J.6
Walter, U.7
-
49
-
-
0032617113
-
1-adrenergic stimulation
-
Iaccarino G, Dolber PC, Lefkowitz RJ, Koch WJ (1999) Beta-adrenergic receptor kinase-1 levels in catecholamine-induced myocardial hypertrophy: Regulation by beta- but not alpha1-adrenergic stimulation. Hypertension 33:396-401. (Pubitemid 29065691)
-
(1999)
Hypertension
, vol.33
, Issue.1 II
, pp. 396-401
-
-
Iaccarino, G.1
Dolber, P.C.2
Lefkowitz, R.J.3
Koch, W.J.4
-
50
-
-
0033535942
-
1-adrenergic receptor transgenic mice
-
DOI 10.1073/pnas.96.12.7059
-
Engelhardt S, Hein L, Wiesmann F, Lohse MJ (1999) Progressive hypertrophy and heart failure in beta1-adrenergic receptor transgenic mice. Proc Natl Acad Sci USA 96: 7059-7064. (Pubitemid 29275009)
-
(1999)
Proceedings of the National Academy of Sciences of the United States of America
, vol.96
, Issue.12
, pp. 7059-7064
-
-
Engelhardt, S.1
Hein, L.2
Wiesmann, F.3
Lohse, M.J.4
-
51
-
-
0037154168
-
Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo
-
Antos CL, et al. (2002) Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo. Proc Natl Acad Sci USA 99:907-912.
-
(2002)
Proc Natl Acad Sci USA
, vol.99
, pp. 907-912
-
-
Antos, C.L.1
-
52
-
-
61749089404
-
Regulator of G protein signaling 2 mediates cardiac compensation to pressure overload and antihypertrophic effects of PDE5 inhibition in mice
-
Takimoto E, et al. (2009) Regulator of G protein signaling 2 mediates cardiac compensation to pressure overload and antihypertrophic effects of PDE5 inhibition in mice. J Clin Invest 119:408-420.
-
(2009)
J Clin Invest
, vol.119
, pp. 408-420
-
-
Takimoto, E.1
-
53
-
-
67349149420
-
Cyclic GMP signaling in cardiovascular pathophysiology and therapeutics
-
Tsai EJ, Kass DA (2009) Cyclic GMP signaling in cardiovascular pathophysiology and therapeutics. Pharmacol Ther 122:216-238.
-
(2009)
Pharmacol Ther
, vol.122
, pp. 216-238
-
-
Tsai, E.J.1
Kass, D.A.2
-
54
-
-
60549098959
-
Ventricular phosphodiesterase-5 expression is increased in patients with advanced heart failure and contributes to adverse ventricular remodeling after myocardial infarction in mice
-
Pokreisz P, et al. (2009) Ventricular phosphodiesterase-5 expression is increased in patients with advanced heart failure and contributes to adverse ventricular remodeling after myocardial infarction in mice. Circulation 119:408-416.
-
(2009)
Circulation
, vol.119
, pp. 408-416
-
-
Pokreisz, P.1
-
55
-
-
0035166852
-
11 in cardiomyocytes
-
DOI 10.1038/nm1101-1236
-
Wettschureck N, et al. (2001) Absence of pressure overload induced myocardial hypertrophy after conditional inactivation of Galphaq/Galpha11 in cardiomyocytes. Nat Med 7:1236-1240. (Pubitemid 33063779)
-
(2001)
Nature Medicine
, vol.7
, Issue.11
, pp. 1236-1240
-
-
Wettschureck, N.1
Rutten, H.2
Zywietz, A.3
Gehring, D.4
Wilkie, T.M.5
Chen, J.6
Chien, K.R.7
Offermanns, S.8
-
56
-
-
0032546640
-
Pressure overload induces cardiac hypertrophy in angiotensin II type 1a receptor knockout mice
-
Harada K, et al. (1998) Pressure overload induces cardiac hypertrophy in angiotensin II type 1A receptor knockout mice. Circulation 97:1952-1959. (Pubitemid 28222190)
-
(1998)
Circulation
, vol.97
, Issue.19
, pp. 1952-1959
-
-
Harada, K.1
-
57
-
-
33845210475
-
Angiotensin II causes hypertension and cardiac hypertrophy through its receptors in the kidney
-
Crowley SD, et al. (2006) Angiotensin II causes hypertension and cardiac hypertrophy through its receptors in the kidney. Proc Natl Acad Sci USA 103:17985-17990.
-
(2006)
Proc Natl Acad Sci USA
, vol.103
, pp. 17985-17990
-
-
Crowley, S.D.1
-
58
-
-
36348933827
-
Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes
-
DOI 10.1074/jbc.M703173200
-
Vandeput F, et al. (2007) Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes. J Biol Chem 282:32749-32757. (Pubitemid 350159317)
-
(2007)
Journal of Biological Chemistry
, vol.282
, Issue.45
, pp. 32749-32757
-
-
Vandeput, F.1
Wolda, S.L.2
Krall, J.3
Hambleton, R.4
Uher, L.5
McCaw, K.N.6
Radwanski, P.B.7
Florio, V.8
Movsesian, M.A.9
-
59
-
-
70349085833
-
CGMP-hydrolytic activity and its inhibition by sildenafil in normal and failing human and mouse myocardium
-
Vandeput F, et al. (2009) cGMP-hydrolytic activity and its inhibition by sildenafil in normal and failing human and mouse myocardium. J Pharmacol Exp Ther 330: 884-891.
-
(2009)
J Pharmacol Exp Ther
, vol.330
, pp. 884-891
-
-
Vandeput, F.1
-
60
-
-
46249094707
-
Role of smooth muscle cGMP/cGKI signaling in murine vascular restenosis
-
Lukowski R, et al. (2008) Role of smooth muscle cGMP/cGKI signaling in murine vascular restenosis. Arterioscler Thromb Vasc Biol 28:1244-1250.
-
(2008)
Arterioscler Thromb Vasc Biol
, vol.28
, pp. 1244-1250
-
-
Lukowski, R.1
-
61
-
-
0344924879
-
Interaction of Smad3 and SRF-associated complex mediates TGF-beta1 signals to regulate SM22 transcription during myofibroblast differentiation
-
DOI 10.1016/j.yjmcc.2003.09.002
-
Qiu P, Feng XH, Li L (2003) Interaction of Smad3 and SRF-associated complex mediates TGF-beta1 signals to regulate SM22 transcription during myofibroblast differentiation. J Mol Cell Cardiol 35:1407-1420. (Pubitemid 37491184)
-
(2003)
Journal of Molecular and Cellular Cardiology
, vol.35
, Issue.12
, pp. 1407-1420
-
-
Qiu, P.1
Feng, X.-H.2
Li, L.3
|