Exercise training as a treatment for heart failure: Potential mechanisms and clinical implications

Journal of Physiology

Volumn 587, Issue 21, 2009, Pages 5011-5013

  Benito, Begoña ^a   Nattel, Stanley ^a  

^a MONTREAL HEART INSTITUTE   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA 2A ADRENERGIC RECEPTOR; ALPHA 2C ADRENERGIC RECEPTOR; CALCINEURIN; DIPEPTIDYL CARBOXYPEPTIDASE; MITOGEN ACTIVATED PROTEIN KINASE P38; NORADRENALIN; PROTEIN KINASE B; SODIUM CALCIUM EXCHANGE PROTEIN; TRANSCRIPTION FACTOR NFAT; CALCIUM CALMODULIN DEPENDENT PROTEIN KINASE II; CALCIUM ION; PHOSPHATIDYLINOSITOL 3 KINASE;

ADRENERGIC STIMULATION; AEROBIC EXERCISE; BETA ADRENERGIC STIMULATION; CLINICAL EFFECTIVENESS; HEART FAILURE; HEART HYPERTROPHY; HEART VENTRICLE REMODELING; HUMAN; KINESIOTHERAPY; NONHUMAN; NORADRENALIN RELEASE; NOTE; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; CARDIAC REMODELING; CARDIOVASCULAR MORTALITY; ENZYME ACTIVITY; EXERCISE; HEART DISEASE; HEART FUNCTION; HEART PROTECTION; HEART VENTRICLE HYPERTROPHY; HOSPITALIZATION; PROTEIN FUNCTION; RANDOMIZED CONTROLLED TRIAL (TOPIC); THERAPY EFFECT;

ANIMALS; CALCINEURIN; CARDIOMEGALY; CARRIER PROTEINS; CYCLOSPORINE; DISEASE MODELS, ANIMAL; ENZYME INHIBITORS; EXERCISE TOLERANCE; HEART FAILURE; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCYTES, CARDIAC; NFATC TRANSCRIPTION FACTORS; PHOSPHOTRANSFERASES (ALCOHOL GROUP ACCEPTOR); PHYSICAL CONDITIONING, ANIMAL; PROTO-ONCOGENE PROTEINS C-AKT; RECEPTORS, ADRENERGIC, ALPHA-2; SIGNAL TRANSDUCTION; VENTRICULAR REMODELING;

EID: 70450219181     PISSN: 00223751     EISSN: 14697793     Source Type: Journal    
DOI: 10.1113/jphysiol.2009.181339     Document Type: Note

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