Neurotoxic lipid peroxidation species formed by ischemic stroke increase injury

Free Radical Biology and Medicine

Volumn 47, Issue 10, 2009, Pages 1422-1431

  Zeiger, Stephanie L H ^a,b   Musiek, Erik S ^a,c   Zanoni, Giuseppe ^d   Vidari, Giovanni ^d   Morrow, Jason D ^a   Milne, Ginger J ^a   McLaughlin, BethAnn ^a,b  

^a VANDERBILT UNIVERSITY   (United States)

^b VANDERBILT UNIVERSITY MEDICAL CENTER   (United States)

^c HOSPITAL OF THE UNIVERSITY OF PENNSYLVANIA   (United States)

^d UNIVERSITY OF PAVIA   (Italy)

Author keywords

Free radicals; Ischemia; Isoprostanes; Neurodegeneration; Reactive oxygen species; Stroke

Indexed keywords

ADENOSINE TRIPHOSPHATE; CALCIUM ION; CYCLOPENTENONE; CYTOCHROME C; GLUCOSE; ISOPROSTANE; OXYGEN; REACTIVE OXYGEN METABOLITE;

AGED; ANIMAL CELL; ARTICLE; BRAIN ISCHEMIA; CALCIUM TRANSPORT; CEREBROVASCULAR ACCIDENT; CONTROLLED STUDY; HUMAN; HUMAN TISSUE; HYPERPOLARIZATION; HYPOXIA; LIPID PEROXIDATION; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRIAL PERMEABILITY; NERVE CELL NECROSIS; NERVE DEGENERATION; NEUROTOXICITY; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL;

ANIMALS; CALCIUM; CELLS, CULTURED; CYTOCHROMES C; DOSE-RESPONSE RELATIONSHIP, DRUG; HUMANS; LIPID PEROXIDATION; MASS SPECTROMETRY; MITOCHONDRIA; NEURONS; PROSTAGLANDINS A; RATS; RATS, SPRAGUE-DAWLEY; REACTIVE OXYGEN SPECIES; REPERFUSION INJURY; STROKE; TIME FACTORS;

EID: 70350126943     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.freeradbiomed.2009.08.011     Document Type: Article

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