Diabetes increases susceptibility of primary cultures of rat proximal tubular cells to chemically induced injury

Toxicology and Applied Pharmacology

Volumn 241, Issue 1, 2009, Pages 1-13

  Zhong, Qing ^a   Terlecky, Stanley R ^a   Lash, Lawrence H ^a  

^a WAYNE STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Diabetes; Glutathione; Mitochondrial function; Oxidative stress; Proximal tubular cells; Rat kidney

Indexed keywords

ACETYLCYSTEINE; ANTIMYCIN A1; CATALASE; GLUTATHIONE; REACTIVE OXYGEN METABOLITE; TERT BUTYL HYDROPEROXIDE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CELL CULTURE; CELL DAMAGE; CELL ISOLATION; CELL MEMBRANE POTENTIAL; CELL PROTECTION; CONTROLLED STUDY; CYTOPLASM; DIABETES MELLITUS; GENETIC SUSCEPTIBILITY; KIDNEY CELL; KIDNEY PROXIMAL TUBULE; MITOCHONDRIAL MEMBRANE; NONHUMAN; OXIDATIVE STRESS; PHENOTYPE; RAT;

ANIMALS; ANTIMYCIN A; ANTIOXIDANTS; APOPTOSIS; BUTANONES; DIABETES MELLITUS, EXPERIMENTAL; DIABETIC NEPHROPATHIES; DISEASE MODELS, ANIMAL; GLUTATHIONE; KIDNEY TUBULES, PROXIMAL; MALE; MEMBRANE POTENTIAL, MITOCHONDRIAL; NECROSIS; OXIDATIVE STRESS; RATS; RATS, SPRAGUE-DAWLEY; REACTIVE OXYGEN SPECIES; STREPTOZOCIN; TERT-BUTYLHYDROPEROXIDE;

RATTUS;

EID: 70350034039     PISSN: 0041008X     EISSN: 10960333     Source Type: Journal    
DOI: 10.1016/j.taap.2009.08.007     Document Type: Article

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