-
1
-
-
38949119408
-
Apoptosis regulators Fas and Bim cooperate in shutdown of chronic immune responses and prevention of autoimmunity
-
Hughes, P. D. et al. Apoptosis regulators Fas and Bim cooperate in shutdown of chronic immune responses and prevention of autoimmunity. Immunity 28, 197-205 (2008)
-
(2008)
Immunity
, vol.28
, pp. 197-205
-
-
Hughes, P.D.1
-
2
-
-
38949209522
-
Combined deficiency of proapoptotic regulators Bim and Fas results in the early onset of systemic autoimmunity
-
Hutcheson, J. et al. Combined deficiency of proapoptotic regulators Bim and Fas results in the early onset of systemic autoimmunity. Immunity 28, 206-217 (2008)
-
(2008)
Immunity
, vol.28
, pp. 206-217
-
-
Hutcheson, J.1
-
3
-
-
38949210228
-
Apoptosis regulators Bim and Fas function concurrently to control autoimmunity and CD81 T cell contraction
-
Weant, A. E. et al. Apoptosis regulators Bim and Fas function concurrently to control autoimmunity and CD81 T cell contraction. Immunity 28, 218-230 (2008)
-
(2008)
Immunity
, vol.28
, pp. 218-230
-
-
Weant, A.E.1
-
4
-
-
0034641898
-
CD95s deadly mission in the immune system
-
Krammer, P. H. CD95s deadly mission in the immune system. Nature 407, 789-795 (2000)
-
(2000)
Nature
, vol.407
, pp. 789-795
-
-
Krammer, P.H.1
-
5
-
-
34247536682
-
The CD95 receptor: Apoptosis revisited
-
Peter, M. E. et al. The CD95 receptor: apoptosis revisited. Cell 129, 447-450 (2007)
-
(2007)
Cell
, vol.129
, pp. 447-450
-
-
Peter, M.E.1
-
6
-
-
0026530828
-
The cDNA structure, expression, and chromosomal assignment of the mouse Fas antigen
-
Watanabe-Fukunaga, R. et al. The cDNA structure, expression, and chromosomal assignment of the mouse Fas antigen. J. Immunol. 148, 1274-1279 (1992)
-
(1992)
J. Immunol.
, vol.148
, pp. 1274-1279
-
-
Watanabe-Fukunaga, R.1
-
7
-
-
0028223847
-
Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand
-
Takahashi, T. et al. Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand. Cell 76, 969-976 (1994)
-
(1994)
Cell
, vol.76
, pp. 969-976
-
-
Takahashi, T.1
-
8
-
-
0029006893
-
Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity
-
Rieux-Laucat, F. et al. Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity. Science 268, 1347-1349 (1995)
-
(1995)
Science
, vol.268
, pp. 1347-1349
-
-
Rieux-Laucat, F.1
-
9
-
-
0029025441
-
Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome.
-
Fisher, G. H. et al. Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome. Cell 81, 935-946 (1995)
-
(1995)
Cell
, vol.81
, pp. 935-946
-
-
Fisher, G.H.1
-
11
-
-
34247382029
-
ADAM10 regulates FasL cell surface expression and modulates FasL-induced cytotoxicity and activation-induced cell death
-
Schulte, M. et al. ADAM10 regulates FasL cell surface expression and modulates FasL-induced cytotoxicity and activation-induced cell death. Cell Death Differ. 14, 1040-1049 (2007)
-
(2007)
Cell Death Differ.
, vol.14
, pp. 1040-1049
-
-
Schulte, M.1
-
12
-
-
0032550366
-
Conversion of membrane-bound Fas(CD95) ligand to its soluble form is associated with downregulation of its proapoptotic activity and loss of liver toxicity
-
Schneider, P. et al. Conversion of membrane-bound Fas(CD95) ligand to its soluble form is associated with downregulation of its proapoptotic activity and loss of liver toxicity. J. Exp. Med. 187, 1205-1213 (1998)
-
(1998)
J. Exp. Med.
, vol.187
, pp. 1205-1213
-
-
Schneider, P.1
-
13
-
-
0031938030
-
Downregulation of Fas ligand by shedding
-
Tanaka, M., Itai, T., Adachi, M. & Nagata, S. Downregulation of Fas ligand by shedding. Nature Med. 4, 31-36 (1998)
-
(1998)
Nature Med.
, vol.4
, pp. 31-36
-
-
Tanaka, M.1
Itai, T.2
Adachi, M.3
Nagata, S.4
-
14
-
-
0034600068
-
Opposing effects of transmembrane and soluble Fas ligand expression on inflammation and tumor cell survival
-
Hohlbaum, A. M., Moe, S. & Marshak-Rothstein, A. Opposing effects of transmembrane and soluble Fas ligand expression on inflammation and tumor cell survival. J. Exp. Med. 191, 1209-1219 (2000)
-
(2000)
J. Exp. Med.
, vol.191
, pp. 1209-1219
-
-
Hohlbaum, A.M.1
Moe, S.2
Marshak-Rothstein, A.3
-
15
-
-
0034733584
-
Fas preassociation required for apoptosis signaling and dominant inhibition by pathogenic mutations
-
Siegel, R. M. et al. Fas preassociation required for apoptosis signaling and dominant inhibition by pathogenic mutations. Science 288, 2354-2357 (2000)
-
(2000)
Science
, vol.288
, pp. 2354-2357
-
-
Siegel, R.M.1
-
16
-
-
0032573581
-
Regulation of the proinflammatory effects of Fas ligand (CD95L)
-
Chen, J.-J., Sun, Y. & Nabel, G. J. Regulation of the proinflammatory effects of Fas ligand (CD95L). Science 282, 1714-1717 (1998)
-
(1998)
Science
, vol.282
, pp. 1714-1717
-
-
Chen, J.-J.1
Sun, Y.2
Nabel, G.J.3
-
17
-
-
62849093368
-
Death receptor signal transducers: Nodes of coordination in immune signaling networks
-
Wilson, N. S., Dixit, V. & Ashkenazi, A. Death receptor signal transducers: nodes of coordination in immune signaling networks. Nature Immunol. 10, 348-355 (2009)
-
(2009)
Nature Immunol.
, vol.10
, pp. 348-355
-
-
Wilson, N.S.1
Dixit, V.2
Ashkenazi, A.3
-
18
-
-
0035861624
-
Non-apoptotic signaling pathways activated by soluble Fas ligand in serum-starved human fibroblasts. Mitogen-activated protein kinases and NFkB- dependent gene expression
-
Ahn, J. H. et al. Non-apoptotic signaling pathways activated by soluble Fas ligand in serum-starved human fibroblasts. Mitogen-activated protein kinases and NFkB- dependent gene expression. J. Biol. Chem. 276, 47100-47106 (2001)
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 47100-47106
-
-
Ahn, J.H.1
-
19
-
-
0037185002
-
A novel signaling mechanism for soluble CD95 ligand. Synergy with anti-CD95 monoclonal antibodies for apoptosis and NF-kB nuclear translocation
-
Xiao, S., Jodo, S., Sung, S. S., Marshak-Rothstein, A. & Ju, S. T. A novel signaling mechanism for soluble CD95 ligand. Synergy with anti-CD95 monoclonal antibodies for apoptosis and NF-kB nuclear translocation. J. Biol. Chem. 277, 50907-50913 (2002)
-
(2002)
J. Biol. Chem.
, vol.277
, pp. 50907-50913
-
-
Xiao, S.1
Jodo, S.2
Sung, S.S.3
Marshak-Rothstein, A.4
Ju, S.T.5
-
20
-
-
34547461299
-
NRAS mutation causes a human autoimmune lymphoproliferative syndrome
-
Oliveira, J. B. et al. NRAS mutation causes a human autoimmune lymphoproliferative syndrome. Proc. Natl Acad. Sci. USA 104, 8953-8958 (2007)
-
(2007)
Proc. Natl Acad. Sci. USA
, vol.104
, pp. 8953-8958
-
-
Oliveira, J.B.1
-
21
-
-
0033487444
-
Genetic susceptibility to spontaneous lupus in mice
-
Kono, D. H. & Theofilopoulos, A. N. Genetic susceptibility to spontaneous lupus in mice. Curr. Dir. Autoimmun. 1, 72-98 (1999)
-
(1999)
Curr. Dir. Autoimmun.
, vol.1
, pp. 72-98
-
-
Kono, D.H.1
Theofilopoulos, A.N.2
-
22
-
-
0028835952
-
Constitutive activation of the Fas ligand gene in mouse lymphoproliferative disorders
-
Watanabe, D., Suda, T., Hashimoto, H. & Nagata, S. Constitutive activation of the Fas ligand gene in mouse lymphoproliferative disorders. EMBO J. 14, 12-18 (1995)
-
(1995)
EMBO J.
, vol.14
, pp. 12-18
-
-
Watanabe, D.1
Suda, T.2
Hashimoto, H.3
Nagata, S.4
-
23
-
-
0027291205
-
Lethal effect of the anti-Fas antibody in mice
-
Ogasawara, J. et al. Lethal effect of the anti-Fas antibody in mice. Nature 364, 806-809 (1993)
-
(1993)
Nature
, vol.364
, pp. 806-809
-
-
Ogasawara, J.1
-
24
-
-
0033592984
-
Activation of Fas by FasL Induces Apoptosis by A Mechanism That Cannot Be Blocked by Bcl-2 or Bcl-xL
-
Huang, D. C. et al. Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-xL. Proc. Natl Acad. Sci. USA 96, 14871-14876 (1999)
-
(1999)
Proc. Natl Acad. Sci. USA
, vol.96
, pp. 14871-14876
-
-
Huang, D.C.1
-
25
-
-
16144363507
-
Lymphocyte apoptosis induced by CD95 (APO-1/Fas) ligandexpressing tumor cells-a mechanism of immune evasion?
-
Strand, S. et al. Lymphocyte apoptosis induced by CD95 (APO-1/Fas) ligandexpressing tumor cells-a mechanism of immune evasion? Nature Med. 2, 1361-1366 (1996)
-
(1996)
Nature Med.
, vol.2
, pp. 1361-1366
-
-
Strand, S.1
-
27
-
-
0019483336
-
Morphologic classification and correlation of incidence of hyperplastic and neoplastic hematopoietic lesions in mice with age
-
Frith, C. H. & Wiley, L. D. Morphologic classification and correlation of incidence of hyperplastic and neoplastic hematopoietic lesions in mice with age. J. Gerontol. 36, 534-545 (1981)
-
(1981)
J. Gerontol.
, vol.36
, pp. 534-545
-
-
Frith, C.H.1
Wiley, L.D.2
-
28
-
-
0031973637
-
Maximal proliferation of cytotoxic T lymphocytes requires reverse signaling through Fas ligand
-
DOI 10.1084/jem.187.1.123
-
Suzuki, I. & Fink, P. J. Maximal proliferation of cytotoxic T lymphocytes requires reverse signaling through Fas ligand. J. Exp. Med. 187, 123-128 (1998) (Pubitemid 28044827)
-
(1998)
Journal of Experimental Medicine
, vol.187
, Issue.1
, pp. 123-128
-
-
Suzuki, I.1
Fink, P.J.2
-
29
-
-
42949162823
-
A new class of reverse signaling costimulators belongs to the TNF family
-
Sun, M. & Fink, P. J. A new class of reverse signaling costimulators belongs to the TNF family. J. Immunol. 179, 4307-4312 (2007)
-
(2007)
J. Immunol.
, vol.179
, pp. 4307-4312
-
-
Sun, M.1
Fink, P.J.2
-
30
-
-
0842321790
-
Complete loss of Fas ligand gene causes massive lymphoproliferation and early death, indicating a residual activity of gld allele
-
Karray, S. et al. Complete loss of Fas ligand gene causes massive lymphoproliferation and early death, indicating a residual activity of gld allele. J. Immunol. 172, 2118-2125 (2004)
-
(2004)
J. Immunol.
, vol.172
, pp. 2118-2125
-
-
Karray, S.1
-
31
-
-
0029566108
-
A cre-transgenic mouse strain for the ubiquitous deletion of loxP-flanked gene segments including deletion in germ cells
-
Schwenk, F., Baron, U. & Rajewsky, K. A cre-transgenic mouse strain for the ubiquitous deletion of loxP-flanked gene segments including deletion in germ cells. Nucleic Acids Res. 23, 5080-5081 (1995)
-
(1995)
Nucleic Acids Res.
, vol.23
, pp. 5080-5081
-
-
Schwenk, F.1
Baron, U.2
Rajewsky, K.3
-
32
-
-
33847364499
-
Patients with chronic pancreatitis have islet progenitor cells in their ducts, but reversal of overt diabetes in NOD mice by anti-CD3 shows no evidence for islet regeneration
-
Phillips, J. M., OReilly, L., Bland, C., Foulis, A. K. & Cooke, A. Patients with chronic pancreatitis have islet progenitor cells in their ducts, but reversal of overt diabetes in NOD mice by anti-CD3 shows no evidence for islet regeneration. Diabetes 56, 634-640 (2007)
-
(2007)
Diabetes
, vol.56
, pp. 634-640
-
-
Phillips, J.M.1
Oreilly, L.2
Bland, C.3
Foulis, A.K.4
Cooke, A.5
-
33
-
-
0035985576
-
Tumours of histiocytes and accessory dendritic cells: An immunohistochemical approach to classification from the International Lymphoma Study Group based on 61 cases
-
Pileri, S. A. et al. Tumours of histiocytes and accessory dendritic cells: an immunohistochemical approach to classification from the International Lymphoma Study Group based on 61 cases. Histopathology 41, 1-29 (2002)
-
(2002)
Histopathology
, vol.41
, pp. 1-29
-
-
Pileri, S.A.1
-
34
-
-
0034927311
-
Fas ligand is targeted to secretory lysosomes via a proline-rich domain in its cytoplasmic tail
-
Blott, E. J., Bossi, G., Clark, R., Zvelebil, M. & Griffiths, G. M. Fas ligand is targeted to secretory lysosomes via a proline-rich domain in its cytoplasmic tail. J. Cell Sci. 114, 2405-2416 (2001)
-
(2001)
J. Cell Sci.
, vol.114
, pp. 2405-2416
-
-
Blott, E.J.1
Bossi, G.2
Clark, R.3
Zvelebil, M.4
Griffiths, G.M.5
-
35
-
-
0038156226
-
CD8a1 mouse spleen dendritic cells do not originate from the CD8 a-dendritic cell subset
-
Naik, S., Vremec, D., Wu, L., OKeeffe, M. & Shortman, K. CD8a1 mouse spleen dendritic cells do not originate from the CD8 a-dendritic cell subset. Blood 102, 601-604 (2003)
-
(2003)
Blood
, vol.102
, pp. 601-604
-
-
Naik, S.1
Vremec, D.2
Wu, L.3
Okeeffe, M.4
Shortman, K.5
-
36
-
-
0028784215
-
Multiple defects in the immune system of Lyn-deficient mice, culminating in autoimmune disease
-
Hibbs, M. L. et al. Multiple defects in the immune system of Lyn-deficient mice, culminating in autoimmune disease. Cell 83, 301-311 (1995)
-
(1995)
Cell
, vol.83
, pp. 301-311
-
-
Hibbs, M.L.1
-
37
-
-
0028262128
-
Determination of lymphocyte division by flow cytometry
-
Lyons, A. B. & Parish, C. R. Determination of lymphocyte division by flow cytometry. J. Immunol. Methods 171, 131-137 (1994)
-
(1994)
J. Immunol. Methods
, vol.171
, pp. 131-137
-
-
Lyons, A.B.1
Parish, C.R.2
-
38
-
-
0014653923
-
Future influenza vaccines and the use of genetic recombinants
-
Kilbourne, E. D. Future influenza vaccines and the use of genetic recombinants. Bull. WHO 41, 643-645 (1969)
-
(1969)
Bull. WHO
, vol.41
, pp. 643-645
-
-
Kilbourne, E.D.1
-
39
-
-
0032100720
-
Virus-specific CD81 T cells in primary and secondary influenza pneumonia
-
Flynn, K. J. et al. Virus-specific CD81 T cells in primary and secondary influenza pneumonia. Immunity 8, 683-691 (1998)
-
(1998)
Immunity
, vol.8
, pp. 683-691
-
-
Flynn, K.J.1
-
40
-
-
0034027174
-
A previously unrecognized H-2D(b) -restricted peptide prominent in the primary influenza A virus-specific CD8(1) T-cell response is much less apparent following secondary challenge
-
Belz, G. T., Xie, W., Altman, J. D. & Doherty, P. C. A previously unrecognized H-2D(b)-restricted peptide prominent in the primary influenza A virus-specific CD8(1) T-cell response is much less apparent following secondary challenge. J. Virol. 74, 3486-3493 (2000)
-
(2000)
J. Virol.
, vol.74
, pp. 3486-3493
-
-
Belz, G.T.1
Xie, W.2
Altman, J.D.3
Doherty, P.C.4
-
41
-
-
0035313650
-
Diversity of epitope and cytokine profiles for primary and secondary influenza a virus-specific CD81 T cell responses
-
Belz, G. T., Xie, W. & Doherty, P. C. Diversity of epitope and cytokine profiles for primary and secondary influenza a virus-specific CD81 T cell responses. J. Immunol. 166, 4627-4633 (2001)
-
(2001)
J. Immunol.
, vol.166
, pp. 4627-4633
-
-
Belz, G.T.1
Xie, W.2
Doherty, P.C.3
|