Genetic disruption of AMPK signaling abolishes both contraction- and insulin-stimulated TBC1D1 phosphorylation and 14-3-3 binding in mouse skeletal muscle

American Journal of Physiology - Endocrinology and Metabolism

Volumn 297, Issue 3, 2009, Pages

  Pehmøller, Christian ^a   Treebak, Jonas T ^a   Birk, Jesper B ^a   Chen, Shuai ^b   MacKintosh, Carol ^b   Hardie, D Grahame ^b   Richter, Erik A ^a   Wojtaszewski, Jørgen F P ^a  

^a UNIVERSITY OF COPENHAGEN   (Denmark)

^b UNIVERSITY OF DUNDEE   (United Kingdom)

Author keywords

5 AMP activated protein kinase; 5 Aminoimidazole 4 carboxamide 1 D ribofuranoside; AMPK kinase dead; Extensor digitorum longus muscle; Wortmannin

Indexed keywords

5 AMINO 4 IMIDAZOLECARBOXAMIDE RIBOSIDE; GUANOSINE TRIPHOSPHATASE ACTIVATING PROTEIN; HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE KINASE; NEUTRAL INSULIN; PROTEIN 14 3 3; PROTEIN TBC1D1; RAB PROTEIN; SERINE; THREONINE; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; EXTENSOR DIGITORUM LONGUS MUSCLE; FEMALE; GENE DISRUPTION; MOUSE; MUSCLE CONTRACTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION; SKELETAL MUSCLE; WILD TYPE;

14-3-3 PROTEINS; AMP-ACTIVATED PROTEIN KINASES; ANIMALS; FEMALE; INSULIN; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MUSCLE CONTRACTION; MUSCLE, SKELETAL; MUTAGENESIS; NUCLEAR PROTEINS; PHOSPHORYLATION; PROTEIN BINDING; PROTEIN-SERINE-THREONINE KINASES; SIGNAL TRANSDUCTION;

EID: 69049099087     PISSN: 01931849     EISSN: 15221555     Source Type: Journal    
DOI: 10.1152/ajpendo.00115.2009     Document Type: Article

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