Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis

Neuroscience

Volumn 163, Issue 1, 2009, Pages 415-429

  Fritsch, B ^a   Qashu, F ^b   Figueiredo, T H ^b   Aroniadou Anderjaska, V ^b   Rogawski, M A ^a,c   Braga, M F M ^b  

^a NATIONAL INSTITUTE OF NEUROLOGICAL DISORDERS AND STROKE   (United States)

^b Uniformed Services University of the Health Sciences   (United States)

^c UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

basolateral amygdala; epileptogenesis; inhibitory synaptic transmission; interneurons; kainate receptors; status epilepticus

Indexed keywords

4 AMINOBUTYRIC ACID; 4 AMINOBUTYRIC ACID A RECEPTOR; 4 AMINOBUTYRIC ACID A RECEPTOR ALPHA1 SUBUNIT; GLUTAMATE DECARBOXYLASE 67; GLUTAMIC ACID; KAINIC ACID; KAINIC ACID RECEPTOR; PROTEIN GLUK1; UNCLASSIFIED DRUG;

4 AMINOBUTYRIC ACID RELEASE; AMYGDALOID NUCLEUS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL DAMAGE; CELL LOSS; CELL SURVIVAL; CONTROLLED STUDY; EPILEPTIC STATE; EPILEPTOGENESIS; GABAERGIC SYSTEM; HISTOPATHOLOGY; IMMUNOHISTOCHEMISTRY; INHIBITORY POSTSYNAPTIC POTENTIAL; INTERNEURON; INTRACELLULAR RECORDING; MALE; NERVE DEGENERATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PYRAMIDAL NERVE CELL; RAT; RECEPTOR UPREGULATION; SYNAPTIC INHIBITION; WESTERN BLOTTING;

AMYGDALA; ANIMALS; CONVULSANTS; DOWN-REGULATION; EPILEPSY; GAMMA-AMINOBUTYRIC ACID; GLUTAMATE DECARBOXYLASE; GLUTAMIC ACID; IMMUNOHISTOCHEMISTRY; INHIBITORY POSTSYNAPTIC POTENTIALS; INTERNEURONS; KAINIC ACID; MALE; NERVE DEGENERATION; NEURAL INHIBITION; ORGANIC CHEMICALS; PATCH-CLAMP TECHNIQUES; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, GABA-A; RECEPTORS, KAINIC ACID; STAINING AND LABELING; STATUS EPILEPTICUS; SYNAPTIC TRANSMISSION;

EID: 68349131631     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2009.06.034     Document Type: Article

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