Chronic lithium treatment up-regulates cell surface NaV1.7 sodium channels via inhibition of glycogen synthase kinase-3 in adrenal chromaffin cells: Enhancement of Na+ influx, Ca2+ influx and catecholamine secretion after lithium withdrawal

Neuropharmacology

Volumn 57, Issue 3, 2009, Pages 311-321

  Yanagita, Toshihiko ^a   Maruta, Toyoaki ^a   Nemoto, Takayuki ^a   Uezono, Yasuhito ^b,c   Matsuo, Kiyotaka ^b   Satoh, Shinya ^a   Yoshikawa, Norie ^a   Kanai, Tasuku ^a   Kobayashi, Hideyuki ^a   Wada, Akihiko ^a  

^a UNIVERSITY OF MIYAZAKI   (Japan)

^b NAGASAKI UNIVERSITY   (Japan)

^c NATIONAL CANCER CENTER RESEARCH INSTITUTE   (Japan)

Author keywords

Adrenal chromaffin cells; Glycogen synthase kinase 3; Lithium; Sodium channel; Transcription; Up regulation

Indexed keywords

2 (2,4 DICHLOROPHENYL) 3 (1 METHYL 3 INDOLYL)MALEIMIDE; 2 (3 CHLORO 4 HYDROXYANILINO) 3 (2 NITROPHENYL)MALEIMIDE; CALCIUM ION; CATECHOLAMINE; CYCLOHEXIMIDE; DACTINOMYCIN; GLYCOGEN SYNTHASE KINASE 3; LITHIUM CHLORIDE; MESSENGER RNA; SAXITOXIN; SODIUM CHANNEL; SODIUM CHANNEL ALPHA SUBUNIT; SODIUM ION; UNCLASSIFIED DRUG; VERATRIDINE; VOLTAGE GATED SODIUM CHANNEL;

ANIMAL CELL; ARTICLE; BINDING AFFINITY; CALCIUM TRANSPORT; CATECHOLAMINE RELEASE; CELL SURFACE; CHROMAFFIN CELL; CHRONIC DRUG ADMINISTRATION; CONTROLLED STUDY; COW; DRUG MECHANISM; ENZYME INHIBITION; GENE EXPRESSION; GENETIC STABILITY; NONHUMAN; PATCH CLAMP; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; SODIUM TRANSPORT; STEADY STATE; TRANSCRIPTION REGULATION;

ANIMALS; ANTIMANIC AGENTS; CALCIUM; CALCIUM CHANNELS; CATECHOLAMINES; CATTLE; CELL MEMBRANE; CELLS, CULTURED; CHROMAFFIN CELLS; GLYCOGEN SYNTHASE KINASE 3; LITHIUM CHLORIDE; MEMBRANE POTENTIALS; PROTEIN STABILITY; RNA, MESSENGER; SODIUM; SODIUM CHANNELS; TRANSCRIPTION, GENETIC;

EID: 67650675027     PISSN: 00283908     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuropharm.2009.05.006     Document Type: Article

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