GAD65-specific cytotoxic T lymphocytes mediate beta-cell death and loss of function

Review of Diabetic Studies

Volumn 6, Issue 1, 2009, Pages 43-53

  Rasche, Sarah ^a   Busick, Rhea Y ^a   Quinn, Anthony ^a  

^a UNIVERSITY OF TOLEDO   (United States)

Author keywords

Antigen; Beta cell; FasL; GAD65; Major histocompatibility complex class I; MHC; Nod.scid; Pathogenesis; T cell; Type 1 diabetes

Indexed keywords

EPITOPE; GLUTAMATE DECARBOXYLASE 65; PERFORIN;

ADOPTIVE TRANSFER; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANTIGEN PRESENTATION; ANTIGEN PRESENTING CELL; ARTICLE; CELL DEATH; CELL SPECIFICITY; COCULTURE; CONTROLLED STUDY; CROSS PRESENTATION; CYTOTOXIC T LYMPHOCYTE; DIABETOGENESIS; INSULIN DEPENDENT DIABETES MELLITUS; INSULIN SYNTHESIS; MOUSE; NONHUMAN; NONOBESE DIABETIC MOUSE; PANCREAS ISLET BETA CELL; PROTEIN EXPRESSION; SCID MOUSE; TARGET CELL;

EID: 67649207839     PISSN: 16136071     EISSN: 16140575     Source Type: Journal    
DOI: 10.1900/RDS.2009.6.43     Document Type: Article

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