Ah receptor represses acute-phase response gene expression without binding to its cognate response element

Laboratory Investigation

Volumn 89, Issue 6, 2009, Pages 695-707

  Patel, Rushang D ^a,b   Murray, Iain A ^a   Flaveny, Colin A ^a,b   Kusnadi, Ann ^a   Perdew, Gary H ^a  

^a PENNSYLVANIA STATE UNIVERSITY   (United States)

^b PENNSYLVANIA STATE UNIVERSITY   (United States)

Author keywords

Acute phase response; Ah receptor; Inflammation; Liver; Serum amyloid; TCDD

Indexed keywords

AMYLOID; AMYLOID A PROTEIN; AROMATIC HYDROCARBON RECEPTOR; C REACTIVE PROTEIN; CARISOPRODOL; CARRIER PROTEIN; CYTOKINE; DNA; HAPTOGLOBIN; HISTONE; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; LIGAND; MESSENGER RNA; PROTEIN C;

ACETYLATION; ACUTE PHASE RESPONSE; ANIMAL CELL; ARTICLE; CELL DEGENERATION; CELL LINE; CELL MATURATION; CELL MEMBRANE PERMEABILITY; CELL MOTION; CONTROLLED STUDY; DNA BINDING; DNA RESPONSIVE ELEMENT; FEMALE; GENE ACTIVATION; GENE EXPRESSION; GENE EXPRESSION REGULATION; GENE REPRESSION; GENE TRANSLOCATION; HUMAN; HUMAN CELL; INFLAMMATION; LIVER; LIVER CELL; MOUSE; MUTANT; NONHUMAN; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN FUNCTION; PROTEIN INDUCTION; PROTEIN SECRETION; STEM CELL;

ACETYLATION; ACTIVE TRANSPORT, CELL NUCLEUS; ACUTE-PHASE REACTION; ANIMALS; ARYL HYDROCARBON RECEPTOR NUCLEAR TRANSLOCATOR; CELL LINE; CELL NUCLEUS; CYTOKINES; FEMALE; GENE EXPRESSION REGULATION; HEPATOCYTES; HUMANS; LIGANDS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MUTATION; NF-KAPPA B; PROTEIN MULTIMERIZATION; RECEPTORS, ARYL HYDROCARBON; RESPONSE ELEMENTS; SERUM AMYLOID A PROTEIN; SIGNAL TRANSDUCTION;

EID: 66549088712     PISSN: 00236837     EISSN: 15300307     Source Type: Journal    
DOI: 10.1038/labinvest.2009.24     Document Type: Article

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