Loss of spinal glycinergic neurons is not necessary for development of neuropathic pain in transgenic mice expressing enhanced green fluorescent protein in glycinergic neurons

Neuroscience

Volumn 159, Issue 3, 2009, Pages 1148-1153

  Hermanns, H ^a   Muth Selbach, U ^a   Lipfert, P ^a   Braun, S ^a   Werdehausen, R ^a   Bauer, I ^a  

^a HEINRICH HEINE UNIVERSITY   (Germany)

Author keywords

cell death; chronic constriction injury; glycinergic transmission; neuropathic pain

Indexed keywords

GLYCINE RECEPTOR; GLYCINE TRANSPORTER 2; GREEN FLUORESCENT PROTEIN;

ALLODYNIA; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BACTERIAL ARTIFICIAL CHROMOSOME; GENE; HYPERALGESIA; IMMUNOREACTIVITY; LUMBAR SPINE; MOUSE; NERVE COMPRESSION; NEUROPATHIC PAIN; NEUROTRANSMISSION; NOCICEPTION; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN EXPRESSION; SHAM PROCEDURE; STEREOLOGY; THORACIC SPINE; TRANSGENIC MOUSE;

ANALYSIS OF VARIANCE; ANIMALS; CELL COUNT; CELL DEATH; CHROMOSOMES, ARTIFICIAL, BACTERIAL; CONSTRICTION, PATHOLOGIC; DISEASE MODELS, ANIMAL; GLYCINE; GLYCINE PLASMA MEMBRANE TRANSPORT PROTEINS; GREEN FLUORESCENT PROTEINS; HYPERALGESIA; LUMBAR VERTEBRAE; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; NEURONS; PAIN; PAIN MEASUREMENT; SPINAL CORD; THORACIC VERTEBRAE;

EID: 62249083925     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2009.01.037     Document Type: Article

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