PP1 inhibition by Aβ peptide as a potential pathological mechanism in Alzheimer's disease

Neurotoxicology and Teratology

Volumn 31, Issue 2, 2009, Pages 85-88

  Vintém, Ana Paula B ^a   Henriques, Ana Gabriela ^a   da Cruz e Silva, Odete A B ^a   da Cruz e Silva, Edgar F ^a  

^a UNIVERSITY OF AVEIRO   (Portugal)

Author keywords

Abeta aggregation; Alzheimer's disease; Amyloid peptide; Phosphatase; phosphorylation; PP1

Indexed keywords

AMYLOID BETA PROTEIN[1-40]; AMYLOID BETA PROTEIN[1-42]; AMYLOID BETA PROTEIN[25-35]; PHOSPHOPROTEIN PHOSPHATASE 1; PRION PROTEIN; SERINE; THREONINE; AMYLOID BETA PROTEIN; AMYLOID BETA PROTEIN (1 42); AMYLOID BETA PROTEIN (25 35); AMYLOID BETA-PROTEIN (1-42); AMYLOID BETA-PROTEIN (25-35); ISOENZYME; PEPTIDE; PEPTIDE FRAGMENT; PRION PROTEIN (106 126); PRION PROTEIN (106-126); PRION PROTEIN (118 135); PRION PROTEIN (118-135);

ALZHEIMER DISEASE; ANIMAL CELL; ARTICLE; CONTROLLED STUDY; DENDRITIC SPINE; DEPRESSION; ENZYME ACTIVITY; ENZYME INHIBITION; EX VIVO STUDY; IN VITRO STUDY; LEARNING; MEMORY; NERVE CELL PLASTICITY; NERVE DEGENERATION; NEUROTOXICITY; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN AGGREGATION; RAT; ANIMAL; CELL STRAIN; DIET; DRUG ANTAGONISM; METABOLISM; NEWBORN; PATHOPHYSIOLOGY; PHYSIOLOGY; PRION;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ANIMALS; ANIMALS, NEWBORN; DIET; ISOENZYMES; PC12 CELLS; PEPTIDE FRAGMENTS; PEPTIDES; PRIONS; PROTEIN PHOSPHATASE 1; RATS;

EID: 61849092013     PISSN: 08920362     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.ntt.2008.11.001     Document Type: Article

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