Keap1/Nrf2 system regulates neuronal survival as revealed through study of keap1 gene-knockout mice

Biochemical and Biophysical Research Communications

Volumn 380, Issue 2, 2009, Pages 298-302

  Satoh, Takumi ^a   Harada, Nobuhiko ^b   Hosoya, Tomonori ^b   Tohyama, Koujiro ^c   Yamamoto, Masayuki ^d   Itoh, Ken ^b  

^a IWATE UNIVERSITY   (Japan)

^b HIROSAKI UNIVERSITY   (Japan)

^c IWATE MEDICAL UNIVERSITY   (Japan)

^d TOHOKU UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

Electrophilic compounds; Keap1; Nrf2; Oxidative stress; Phase 2 enzymes

Indexed keywords

GLUTAMIC ACID; KELCH LIKE ECH ASSOCIATED PROTEIN 1; ROTENONE; TRANSCRIPTION FACTOR NRF2;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; BRAIN NERVE CELL; BRAIN NUCLEUS; CELL SURVIVAL; GENE DELETION; IMMUNOFLUORESCENCE; KNOCKOUT MOUSE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; WILD TYPE;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; ANIMALS; CELL NUCLEUS; CELL SURVIVAL; CYTOSKELETAL PROTEINS; GLUTAMIC ACID; MICE; MICE, KNOCKOUT; NEURONS; NF-E2-RELATED FACTOR 2; OXIDATIVE STRESS; PROTEIN TRANSPORT; ROTENONE;

MUS;

EID: 60349106093     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2009.01.063     Document Type: Article

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