Akt activation prevents the force drop induced by eccentric contractions in dystrophin-deficient skeletal muscle

Human Molecular Genetics

Volumn 17, Issue 23, 2008, Pages 3686-3696

  Blaauw, Bert ^a,b   Mammucari, Cristina ^a,b   Toniolo, Luana ^a   Agatea, Lisa ^a,b   Abraham, Reimar ^b   Sandri, Marco ^a,b,c   Reggiani, Carlo ^a   Schiaffino, Stefano ^a,b  

^a UNIVERSITY OF PADOVA   (Italy)

^b VENETIAN INSTITUTE OF MOLECULAR MEDICINE   (Italy)

^c DULBECCO TELETHON INSTITUTE   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

DYSFERLIN; DYSTROPHIN; PROTEIN KINASE B; RAPAMYCIN; TAMOXIFEN; UTROPHIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DRUG MECHANISM; DUCHENNE MUSCULAR DYSTROPHY; ECCENTRIC MUSCLE CONTRACTION; ENZYME ACTIVATION; ENZYME ACTIVITY; GENE CONTROL; GENETIC SUSCEPTIBILITY; IN VIVO STUDY; MICROARRAY ANALYSIS; MOUSE; MUSCLE CELL; MUSCLE FORCE; MUSCLE HYPERTROPHY; MUSCLE ISOMETRIC CONTRACTION; MUSCLE STRETCHING; NONHUMAN; POLYMERASE CHAIN REACTION; PRIMARY PREVENTION; PRIORITY JOURNAL; PROTEIN FUNCTION; STRAIN DIFFERENCE; TRANSGENICS; UPREGULATION; WILD TYPE;

ANIMALS; DYSTROPHIN; GENE EXPRESSION; HUMANS; HYPERTROPHY; MICE; MICE, INBRED MDX; MUSCLE CONTRACTION; MUSCLE, SKELETAL; MUSCULAR DYSTROPHY, DUCHENNE; PROTO-ONCOGENE PROTEINS C-AKT; TRANSCRIPTIONAL ACTIVATION;

MUS;

EID: 56049126195     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/ddn264     Document Type: Article

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