Pim-1 kinase antagonizes aspects of myocardial hypertrophy and compensation to pathological pressure overload

Proceedings of the National Academy of Sciences of the United States of America

Volumn 105, Issue 37, 2008, Pages 13889-13894

  Muraski, John A ^a   Fischer, Kimberlee M ^a   Wu, Weitao ^a   Cottage, Christopher T ^a   Quijada, Pearl ^a   Mason, Matt ^a   Din, Shabana ^a   Gude, Natalie ^a   Alvarez Jr , Roberto ^a   Rota, Marcello ^b   Kajstura, Jan ^b   Wang, Zeping ^c   Schaefer, Erik ^c   Chen, Xiongen ^e   MacDonnel, Scott ^e   Magnuson, Nancy ^d   Houser, Stephen R ^e   Anversa, Piero ^b   Sussman, Mark A ^a  

^a SAN DIEGO STATE UNIVERSITY   (United States)

^b BRIGHAM AND WOMEN S HOSPITAL   (United States)

^c INVITROGEN CORPORATION   (United States)

^d WASHINGTON STATE UNIVERSITY   (United States)

^e TEMPLE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BROXURIDINE; PROTEIN KINASE PIM 1;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL CULTURE; CONGESTIVE CARDIOMYOPATHY; CONTROLLED STUDY; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART PROTECTION; HEART VENTRICLE HYPERTROPHY; HEMODYNAMICS; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; TRANSGENIC MOUSE; WILD TYPE;

ANIMALS; ANIMALS, GENETICALLY MODIFIED; AORTA; APOPTOSIS; CARDIOMEGALY; CELLS, CULTURED; ENDOTHELIN-1; FIBROSIS; MUSCLE CONTRACTION; PROTO-ONCOGENE PROTEINS C-PIM-1; RATS;

MUS; MUS MUSCULUS; RATTUS;

EID: 52949116043     PISSN: 00278424     EISSN: 10916490     Source Type: Journal    
DOI: 10.1073/pnas.0709135105     Document Type: Article

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