Differential antiproliferative mechanisms of novel derivative of benzimidazo[1,2-α]quinoline in colon cancer cells depending on their p53 status

Molecular Cancer Therapeutics

Volumn 7, Issue 7, 2008, Pages 2121-2132

  Sedic, Mirela ^a   Poznic, Miroslav ^a   Gehrig, Peter ^c   Scott, Mike ^c   Schlapbach, Ralph ^c   Hranjec, Marijana ^b   Karminski Zamola, Grace ^b   Pavelic, Kresimir ^a   Pavelic, Sandra Kraljevic ^a  

^a RUDER BO KOVI INSTITUTE   (Croatia)

^b UNIVERSITY OF ZAGREB   (Croatia)

^c FUNCTIONAL GENOMICS CENTER ZURICH   (Switzerland)

Author keywords

[No Author keywords available]

Indexed keywords

BENZIMIDAZO[1,2 ALPHA]QUINOLINE; DNA; PROTEIN P53; QUINOLINE DERIVATIVE; UNCLASSIFIED DRUG; BENZIMIDAZOLE DERIVATIVE; QUINOLINE; TUMOR PROTEIN;

ANTINEOPLASTIC ACTIVITY; APOPTOSIS; ARTICLE; BIOGENESIS; CANCER CELL CULTURE; CANCER GROWTH; CELL CYCLE G1 PHASE; CELL CYCLE M PHASE; CELL CYCLE REGULATION; CELL CYCLE S PHASE; CELL DEATH; CELL PROLIFERATION; CELL STRAIN HCT116; COLORECTAL CARCINOMA; DNA DAMAGE; DRUG INHIBITION; HUMAN; HUMAN CELL; MASS SPECTROMETRY; METASTASIS INHIBITION; MITOSIS RATE; NUCLEOTIDE SEQUENCE; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEOMICS; UNINDEXED SEQUENCE; UPREGULATION; WILD TYPE; BIOSYNTHESIS; CELL CYCLE; COLON TUMOR; DRUG EFFECT; METABOLISM; PATHOLOGY; TUMOR CELL LINE; WESTERN BLOTTING;

BENZIMIDAZOLES; BLOTTING, WESTERN; CELL CYCLE; CELL DEATH; CELL LINE, TUMOR; CELL PROLIFERATION; COLONIC NEOPLASMS; DNA, NEOPLASM; HUMANS; NEOPLASM PROTEINS; PROTEOMICS; QUINOLINES; TUMOR SUPPRESSOR PROTEIN P53;

EID: 51049109896     PISSN: 15357163     EISSN: None     Source Type: Journal    
DOI: 10.1158/1535-7163.MCT-07-2261     Document Type: Article

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