메뉴 건너뛰기




Volumn 101, Issue 4, 2004, Pages 912-917

Influence of propofol on neuronal damage and apoptotic factors after incomplete cerebral ischemia and reperfusion in rats: A long-term observation

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; FENTANYL; NITROUS OXIDE; OXYGEN; PROPOFOL; PROTEIN BAX; PROTEIN BCL 2; PROTEIN MDM2; PROTEIN P53;

EID: 4644258294     PISSN: 00033022     EISSN: None     Source Type: Journal    
DOI: 10.1097/00000542-200410000-00016     Document Type: Article
Times cited : (116)

References (27)
  • 1
    • 0036237417 scopus 로고    scopus 로고
    • Propofol anesthesia compared to awake reduces infarct size in rats
    • Gelb AW, Bayona NA, Wilson JX, Cechetto DF: Propofol anesthesia compared to awake reduces infarct size in rats. 2002; ANESTHESIOLOGY 96:1183-90
    • (2002) Anesthesiology , vol.96 , pp. 1183-1190
    • Gelb, A.W.1    Bayona, N.A.2    Wilson, J.X.3    Cechetto, D.F.4
  • 2
    • 0026541916 scopus 로고
    • The effect of propofol on brain electrical activity, neurologic outcome, and neuronal damage following incomplete ischemia in rats
    • Kochs E, Hoffman WE, Werner C, Thomas C, Albrecht RF, Schulte am Esch J: The effect of propofol on brain electrical activity, neurologic outcome, and neuronal damage following incomplete ischemia in rats. ANESTHESIOLOGY 1992; 76:245-52
    • (1992) Anesthesiology , vol.76 , pp. 245-252
    • Kochs, E.1    Hoffman, W.E.2    Werner, C.3    Thomas, C.4    Albrecht, R.F.5    Schulte Am Esch, J.6
  • 4
    • 0031770691 scopus 로고    scopus 로고
    • A histochemical examination of the staining of kainate-induced neuronal degeneration by anionic dyes
    • Kiernan JA, Macpherson CM, Price A, Sun T: A histochemical examination of the staining of kainate-induced neuronal degeneration by anionic dyes. Biotech Histochem 1998; 73:244-54
    • (1998) Biotech Histochem , vol.73 , pp. 244-254
    • Kiernan, J.A.1    Macpherson, C.M.2    Price, A.3    Sun, T.4
  • 6
    • 0021260484 scopus 로고
    • Delayed neuronal death in the rat hippocampus following transient forebrain ischemia
    • Kirino T, Tamura A, Sano K: Delayed neuronal death in the rat hippocampus following transient forebrain ischemia. Acta Neuropathol 1984; 64:139-47
    • (1984) Acta Neuropathol , vol.64 , pp. 139-147
    • Kirino, T.1    Tamura, A.2    Sano, K.3
  • 7
    • 0020067965 scopus 로고
    • Temporal profile of neuronal damage in a model of transient forebrain ischemia
    • Pulsinelli WA, Brierley JB, Plum F: Temporal profile of neuronal damage in a model of transient forebrain ischemia. Ann Neurol 1982; 11:491-8
    • (1982) Ann Neurol , vol.11 , pp. 491-498
    • Pulsinelli, W.A.1    Brierley, J.B.2    Plum, F.3
  • 8
    • 0022333159 scopus 로고
    • Selective neuronal vulnerability: Morphological and molecular characteristics
    • Pulsinelli W: Selective neuronal vulnerability: Morphological and molecular characteristics. Prog Brain Res 1985; 63:29-37
    • (1985) Prog Brain Res , vol.63 , pp. 29-37
    • Pulsinelli, W.1
  • 9
    • 0037384802 scopus 로고    scopus 로고
    • Signaling of cell death and cell survival following focal cerebral ischemia: Life and death struggle in the penumbra
    • Ferrer I, Planas AM: Signaling of cell death and cell survival following focal cerebral ischemia: Life and death struggle in the penumbra. J Neuropathol Exp Neurol 2003; 62:329-39
    • (2003) J Neuropathol Exp Neurol , vol.62 , pp. 329-339
    • Ferrer, I.1    Planas, A.M.2
  • 10
    • 0035045290 scopus 로고    scopus 로고
    • Intracellular Bax translocation after transient cerebral ischemia: Implications for a role of the mitochondrial apoptotic signaling pathway in ischemic neuronal death
    • Cao G, Minami M, Pei W, Yan C, Chen D, O'Horo C, Graham SH, Chen J: Intracellular Bax translocation after transient cerebral ischemia: Implications for a role of the mitochondrial apoptotic signaling pathway in ischemic neuronal death. J Cereb Blood Flow Metab 2001; 21:321-33
    • (2001) J Cereb Blood Flow Metab , vol.21 , pp. 321-333
    • Cao, G.1    Minami, M.2    Pei, W.3    Yan, C.4    Chen, D.5    O'Horo, C.6    Graham, S.H.7    Chen, J.8
  • 12
    • 0000064578 scopus 로고    scopus 로고
    • Altered expression of Bcl-2, Bcl-x, Bax, and c-Fos colocalizes with DNA fragmentation and ischemic cell damage following cerebral artery occlusion in rats
    • Gillardon F, Lenz C, Waschke KF, Krajewski S, Reed JC, Zimmermann M, Kuschinsky W: Altered expression of Bcl-2, Bcl-x, Bax, and c-Fos colocalizes with DNA fragmentation and ischemic cell damage following cerebral artery occlusion in rats. Molecular Brain Res 1996; 40:254-60
    • (1996) Molecular Brain Res , vol.40 , pp. 254-260
    • Gillardon, F.1    Lenz, C.2    Waschke, K.F.3    Krajewski, S.4    Reed, J.C.5    Zimmermann, M.6    Kuschinsky, W.7
  • 13
    • 0037707407 scopus 로고    scopus 로고
    • The effect of hypothermia on the expression of the apoptosis-regulating protein Bax after incomplete cerebral ischemia and reperfusion in rats
    • Eberspächer E, Werner C, Engelhard K, Pape M, Gelb AW, Hutzier P, Henke J, Kochs E: The effect of hypothermia on the expression of the apoptosis-regulating protein Bax after incomplete cerebral ischemia and reperfusion in rats. J Neurosurg Anesthesiol 2003; 15:200-8
    • (2003) J Neurosurg Anesthesiol , vol.15 , pp. 200-208
    • Eberspächer, E.1    Werner, C.2    Engelhard, K.3    Pape, M.4    Gelb, A.W.5    Hutzier, P.6    Henke, J.7    Kochs, E.8
  • 14
    • 0027166048 scopus 로고
    • Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death
    • Oltavi ZN. Milliman CL, Korsmeyer SJ: Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death. Cell 1993; 74:609-19
    • (1993) Cell , vol.74 , pp. 609-619
    • Oltavi, Z.N.1    Milliman, C.L.2    Korsmeyer, S.J.3
  • 15
    • 0032997952 scopus 로고    scopus 로고
    • Bcl-2 transduction, using a herpes simplex virus amplicon, protects hippocampal neurons from transient global ischemia
    • Antonawich FJ. Federoff HJ, Davis JN: Bcl-2 transduction, using a herpes simplex virus amplicon, protects hippocampal neurons from transient global ischemia. Exp Neurol 1999; 156:130-7
    • (1999) Exp Neurol , vol.156 , pp. 130-137
    • Antonawich, F.J.1    Federoff, H.J.2    Davis, J.N.3
  • 18
    • 0031776662 scopus 로고    scopus 로고
    • Increased mdm-2 expression in rat brain after transient middle cerebral artery occlusion
    • Tu Y, Hou S-T, Huang Z, Robertson GS, MacManus JP: Increased mdm-2 expression in rat brain after transient middle cerebral artery occlusion. J Cereb Blood Flow Metab 1998; 18:658-69
    • (1998) J Cereb Blood Flow Metab , vol.18 , pp. 658-669
    • Tu, Y.1    Hou, S.-T.2    Huang, Z.3    Robertson, G.S.4    MacManus, J.P.5
  • 19
    • 3342941800 scopus 로고    scopus 로고
    • Sevoflurane and propofol influence the expression of apoptosis-regulating proteins after cerebral ischaemia and reperfusion in rats
    • Engelhard K, Werner C, Eberspacher E, Pape M, Blobner M, Hutzier P, Kochs E: Sevoflurane and propofol influence the expression of apoptosis-regulating proteins after cerebral ischaemia and reperfusion in rats. Eur J Anaesthesiol 2004; 21:1-8
    • (2004) Eur J Anaesthesiol , vol.21 , pp. 1-8
    • Engelhard, K.1    Werner, C.2    Eberspacher, E.3    Pape, M.4    Blobner, M.5    Hutzier, P.6    Kochs, E.7
  • 20
    • 0028263192 scopus 로고
    • p53-immunoreactive protein and p53 mRNA expression after transient middle cerebral artery occlusion in rats
    • Li Y, Chopp M, Zhang ZG, Zaloga C, Niewenhuis L, Gautam S: p53-immunoreactive protein and p53 mRNA expression after transient middle cerebral artery occlusion in rats. Stroke 1994; 25:849-56
    • (1994) Stroke , vol.25 , pp. 849-856
    • Li, Y.1    Chopp, M.2    Zhang, Z.G.3    Zaloga, C.4    Niewenhuis, L.5    Gautam, S.6
  • 21
    • 0037067518 scopus 로고    scopus 로고
    • Implication of p53 and caspase-3 in kainic acid but not in N-methyl-D-aspartic acid-induced apoptosis in organotypic hippocampal mouse cultures
    • Djebaili M, DeBock F, Bailie V, Bockaert J, Rondouin G: Implication of p53 and caspase-3 in kainic acid but not in N-methyl-D-aspartic acid-induced apoptosis in organotypic hippocampal mouse cultures. Neurosci Let 2002; 327:1-4
    • (2002) Neurosci Let , vol.327 , pp. 1-4
    • Djebaili, M.1    DeBock, F.2    Bailie, V.3    Bockaert, J.4    Rondouin, G.5
  • 24
    • 0036479012 scopus 로고    scopus 로고
    • Apoptosis-inducing factor (AIF): A novel caspase-independent death effector released from mitochondria
    • Cande C, Cohen I, Daugas E, Ravagnan L, Larochette N, Zamzami N, Kroemer G: Apoptosis-inducing factor (AIF): A novel caspase-independent death effector released from mitochondria. Biochimie 2002; 84:215-22
    • (2002) Biochimie , vol.84 , pp. 215-222
    • Cande, C.1    Cohen, I.2    Daugas, E.3    Ravagnan, L.4    Larochette, N.5    Zamzami, N.6    Kroemer, G.7
  • 25
    • 0037302813 scopus 로고    scopus 로고
    • 2-agonist dexmedetomidine and the NMDA-antagonist S(+)-ketamine on the expression of apoptosts-regulating proteins after incomplete cerebral ischemia and reperfusion in rats
    • 2-agonist dexmedetomidine and the NMDA-antagonist S(+)-ketamine on the expression of apoptosts-regulating proteins after incomplete cerebral ischemia and reperfusion in rats. Anesth Analg 2003; 96:524-31
    • (2003) Anesth Analg , vol.96 , pp. 524-531
    • Engelhard, K.1    Werner, C.2    Eberspächer, E.3    Bachl, M.4    Blobner, M.5    Hildt, E.6    Hutzier, R.7    Kochs, E.8
  • 27
    • 0036310780 scopus 로고    scopus 로고
    • Tolerance against ischemic neuronal injury can be induced by volatile anesthetics and is inducible NO synthase dependent
    • Kapinya KJ, Lowl D, Futterer C, Maurer M, Waschke KF, Isaev NK, Dirnagl U: Tolerance against ischemic neuronal injury can be induced by volatile anesthetics and is inducible NO synthase dependent. Stroke 2002; 33:1889-98
    • (2002) Stroke , vol.33 , pp. 1889-1898
    • Kapinya, K.J.1    Lowl, D.2    Futterer, C.3    Maurer, M.4    Waschke, K.F.5    Isaev, N.K.6    Dirnagl, U.7


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.