Reactive oxygen species induced by the deletion of peroxiredoxin II (PrxII) increases the number of thymocytes resulting in the enlargement of PrxII-null thymus

European Journal of Immunology

Volumn 34, Issue 8, 2004, Pages 2119-2128

  Moon, Eun Yi ^a   Han, Ying Hao ^a   Lee, Dong Seok ^a   Han, Yong Mahn ^a   Yu, Dae Yeul ^a  

^a Korea Research Institute of Bioscience and Biotechnology (KRIBB)   (South Korea)

Author keywords

Null mice; Peroxiredoxin II; ROS; Thymocyte

Indexed keywords

CD4 ANTIGEN; CD8 ANTIGEN; CONCANAVALIN A; DEXAMETHASONE; GLUCOCORTICOID RECEPTOR; OKT 3; PEROXIREDOXIN; PEROXIREDOXIN II; PHYTOHEMAGGLUTININ; REACTIVE OXYGEN METABOLITE; UNCLASSIFIED DRUG; PEROXIDASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL FOOD; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL DEATH; CELL MATURATION; CELL SELECTION; CELL STIMULATION; CONTROLLED STUDY; DIET RESTRICTION; DIPLOIDY; GENE DELETION; IMMUNOREGULATION; IN VIVO STUDY; LYMPHOCYTE COUNT; LYMPHOCYTE PROLIFERATION; LYMPHOID TISSUE; NONHUMAN; PERIPHERAL BLOOD MONONUCLEAR CELL; PRIORITY JOURNAL; T LYMPHOCYTE; T LYMPHOCYTE ACTIVATION; THYMOCYTE; THYMUS HYPERPLASIA; WILD TYPE; ANIMAL; CELL DIVISION; CYTOLOGY; GENETICS; HUMAN; METABOLISM; MOUSE; PATHOLOGY; PHYSIOLOGY; THYMUS;

ANIMALS; APOPTOSIS; CELL DIVISION; GENE DELETION; HUMANS; MICE; PEROXIDASES; REACTIVE OXYGEN SPECIES; RECEPTORS, GLUCOCORTICOID; T-LYMPHOCYTES; THYMUS GLAND;

EID: 4444246996     PISSN: 00142980     EISSN: None     Source Type: Journal    
DOI: 10.1002/eji.200424962     Document Type: Article

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