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Volumn 586, Issue 10, 2008, Pages 2427-2428
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Mitochondria, oxidative stress and PARP-1 network: A new target for neuroprotective effects of tetracyclines?
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Author keywords
[No Author keywords available]
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Indexed keywords
MINOCYCLINE;
NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE 1;
REACTIVE OXYGEN METABOLITE;
TETRACYCLINE DERIVATIVE;
CYTOCHROME C;
N METHYL DEXTRO ASPARTIC ACID;
PROTEIN BCL 2;
ARTICLE;
BRAIN INFARCTION;
CELL TRANSPORT;
DEGENERATIVE DISEASE;
DNA DAMAGE;
DRUG MECHANISM;
ENZYME INHIBITION;
EXCITOTOXICITY;
HUMAN;
HUNTINGTON CHOREA;
MITOCHONDRIAL RESPIRATION;
MULTIPLE SCLEROSIS;
NERVE CELL NECROSIS;
NEUROPATHOLOGY;
NEUROPROTECTION;
NONHUMAN;
OXIDATIVE STRESS;
PARKINSON DISEASE;
PATHOPHYSIOLOGY;
PRIORITY JOURNAL;
SIGNAL TRANSDUCTION;
STROKE;
BIOACCUMULATION;
BRAIN HEMORRHAGE;
BRAIN ISCHEMIA;
CELL DEATH;
CELL MEMBRANE DEPOLARIZATION;
ENZYME ACTIVATION;
EXTRACELLULAR CALCIUM;
MITOCHONDRIAL PERMEABILITY;
MITOCHONDRION;
PROTEIN SECRETION;
PROTEIN TARGETING;
UPREGULATION;
ANIMALS;
DRUG DELIVERY SYSTEMS;
HUMANS;
MITOCHONDRIA;
NEUROPROTECTIVE AGENTS;
OXIDATIVE STRESS;
POLY(ADP-RIBOSE) POLYMERASES;
TETRACYCLINES;
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EID: 43749122572
PISSN: 00223751
EISSN: 14697793
Source Type: Journal
DOI: 10.1113/jphysiol.2008.152819 Document Type: Article |
Times cited : (13)
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References (5)
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