Reduction of myeloid suppressor cell derived nitric oxide provides a mechanistic basis of lead enhancement of alloreactive CD4+ T cell proliferation

Toxicology and Applied Pharmacology

Volumn 229, Issue 2, 2008, Pages 135-145

  Farrer, David G ^a   Hueber, Sara ^a   Laiosa, Michael D ^a   Eckles, Kevin G ^a   McCabe Jr , Michael J ^a  

^a UNIVERSITY OF ROCHESTER SCHOOL OF MEDICINE AND DENTISTRY   (United States)

Author keywords

Allogenicity; Immune modulators; Immunotoxicity; Metals; Reactive nitrogen intermediates

Indexed keywords

CD11B ANTIGEN; GAMMA INTERFERON; GLYCOPROTEIN P 15095; LEAD; NITRIC OXIDE; NITRIC OXIDE SYNTHASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; ATTENUATION; BONE MARROW CELL; CD4+ T LYMPHOCYTE; CELL CYCLE; CELL CYCLE REGULATION; CELL PROLIFERATION; FEMALE; IMMUNOTOXICITY; MOUSE; NONHUMAN; SUPPRESSOR CELL; TARGET CELL;

ANIMALS; BASE SEQUENCE; CD4-POSITIVE T-LYMPHOCYTES; CELL LINE; CELL PROLIFERATION; DNA PRIMERS; INTERFERON TYPE II; LEAD; LIPOPOLYSACCHARIDES; MICE; MICE, INBRED C57BL; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE II; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;

MUS;

EID: 43449085183     PISSN: 0041008X     EISSN: 10960333     Source Type: Journal    
DOI: 10.1016/j.taap.2007.12.011     Document Type: Article

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