First molecular evidence that inositol trisphosphate signaling contributes to infarct size reduction with preconditioning

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 291, Issue 4, 2006, Pages

  Przyklenk, Karin ^a   Maynard, Michelle ^a   Whittaker, Peter ^a  

^a UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL   (United States)

Author keywords

Inositol 1,4,5 trisphosphate; Myocardial infarction; Myocardial ischemia; Signal transduction

Indexed keywords

D MYO INOSITOL 1,4,5 TRIPHOSPHATE; DIAZOXIDE; INOSITOL 1,4,5 TRISPHOSPHATE; INOSITOL 1,4,5 TRISPHOSPHATE RECEPTOR; INOSITOL TRISPHOSPHATE; POTASSIUM CHANNEL; PROTEIN KINASE C; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DRUG EFFECT; HEART INFARCTION SIZE; HEART LEFT VENTRICLE; HEART MUSCLE FIBROSIS; HEART MUSCLE NECROSIS; HEART PROTECTION; HEART WEIGHT; ISCHEMIC PRECONDITIONING; MOLECULAR BIOLOGY; MOUSE; MUSCLE CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN EXPRESSION; REPERFUSION;

ANIMALS; CALCIUM CHANNELS; DIAZOXIDE; GENE EXPRESSION REGULATION; HEART VENTRICLES; INOSITOL 1,4,5-TRISPHOSPHATE; ISCHEMIC PRECONDITIONING, MYOCARDIAL; MEMBRANE GLYCOPROTEINS; MICE; MICE, INBRED C57BL; MICE, MUTANT STRAINS; MYOCARDIAL INFARCTION; MYOCARDIUM; PROTEIN KINASE C; RECEPTORS, CYTOPLASMIC AND NUCLEAR; SIGNAL TRANSDUCTION; VASODILATOR AGENTS;

EID: 33749321920     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00313.2006     Document Type: Article

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