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Volumn 105, Issue 21, 2002, Pages 2543-2548
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Intracellular Na+ concentration is elevated in heart failure but Na/K pump function is unchanged
a a a a a |
Author keywords
Calcium; Heart failure; Sodium
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Indexed keywords
ADENOSINE TRIPHOSPHATASE (POTASSIUM SODIUM);
BENZOFURAN DERIVATIVE;
CALCIUM;
ISOPHTHALIC ACID;
SODIUM;
TETRODOTOXIN;
ACTION POTENTIAL;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
CALCIUM TRANSPORT;
CELL STIMULATION;
CONTROLLED STUDY;
ELECTRIC ACTIVITY;
HEART FAILURE;
HEART MUSCLE CELL;
HEART MUSCLE CONTRACTILITY;
NONHUMAN;
PRIORITY JOURNAL;
RABBIT;
SODIUM CALCIUM EXCHANGE;
SODIUM CELL LEVEL;
SODIUM TRANSPORT;
NON-PROGRAMMATIC;
ACTION POTENTIALS;
ANIMALS;
CALCIUM;
CELL SEPARATION;
DIASTOLE;
DISEASE MODELS, ANIMAL;
ELECTRIC STIMULATION;
HEART FAILURE, CONGESTIVE;
INTRACELLULAR FLUID;
MEMBRANE POTENTIALS;
MYOCARDIAL CONTRACTION;
MYOCARDIUM;
NA(+)-K(+)-EXCHANGING ATPASE;
NICKEL;
PATCH-CLAMP TECHNIQUES;
RABBITS;
SODIUM;
SODIUM CHANNEL BLOCKERS;
SODIUM CHANNELS;
SODIUM-CALCIUM EXCHANGER;
STROPHANTHIDIN;
TETRODOTOXIN;
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EID: 0037188622
PISSN: 00097322
EISSN: None
Source Type: Journal
DOI: 10.1161/01.CIR.0000016701.85760.97 Document Type: Article |
Times cited : (280)
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References (29)
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