-
1
-
-
0035057159
-
+ exchanger: A potential therapeutic target for the prevention of myocardial ischaemic and reperfusion injury and attenuation of postinfarction heart failure
-
+ exchanger: a potential therapeutic target for the prevention of myocardial ischaemic and reperfusion injury and attenuation of postinfarction heart failure. Drugs 2001;61:375-389.
-
(2001)
Drugs
, vol.61
, pp. 375-389
-
-
Karmazyn, M.1
Sostaric, J.V.2
Gan, X.T.3
-
3
-
-
0037028890
-
Na(+)/H(+) exchange inhibitors for cardioprotective therapy: Progress, problems and prospects
-
Avkiran M, Marber MS. Na(+)/H(+) exchange inhibitors for cardioprotective therapy: progress, problems and prospects. J Am Coll Cardiol 2002;39:747-753.
-
(2002)
J Am Coll Cardiol
, vol.39
, pp. 747-753
-
-
Avkiran, M.1
Marber, M.S.2
-
4
-
-
0032789045
-
If ischaemic preconditioning is the gold standard, has a platinum standard of cardioprotection arrived? Comparison with NHE inhibition
-
Gumina RJ, Gross GJ. If ischaemic preconditioning is the gold standard, has a platinum standard of cardioprotection arrived? Comparison with NHE inhibition. J Thromb Thrombolysis 1999;8:39-44.
-
(1999)
J Thromb Thrombolysis
, vol.8
, pp. 39-44
-
-
Gumina, R.J.1
Gross, G.J.2
-
5
-
-
0037372388
-
Effective protection by NHE-1 inhibition in ischaemic and reperfused heart under preconditioning blockade
-
Haist JV, Hirst CN, Karmazyn M. Effective protection by NHE-1 inhibition in ischaemic and reperfused heart under preconditioning blockade. Am J Physiol Heart Circ Physiol 2003;284:H798-H803.
-
(2003)
Am J Physiol Heart Circ Physiol
, vol.284
-
-
Haist, J.V.1
Hirst, C.N.2
Karmazyn, M.3
-
6
-
-
34250729007
-
Mitochondrial permeability transition pore opening as an endpoint to initiate cell death and as a putative target for cardioprotection
-
Javadov S, Karmazyn M. Mitochondrial permeability transition pore opening as an endpoint to initiate cell death and as a putative target for cardioprotection. Cell Physiol Biochem 2007;20:1-22.
-
(2007)
Cell Physiol Biochem
, vol.20
, pp. 1-22
-
-
Javadov, S.1
Karmazyn, M.2
-
7
-
-
33646060099
-
Mitochondria and ischaemia-reperfusion injury of the heart: Fixing a hole
-
Di Lisa F, Bernardi P. Mitochondria and ischaemia-reperfusion injury of the heart: fixing a hole. Cardiovasc Res 2006;70:191-199.
-
(2006)
Cardiovasc Res
, vol.70
, pp. 191-199
-
-
Di Lisa, F.1
Bernardi, P.2
-
8
-
-
11144287220
-
Apoptosis of ventricular myocytes: A means to an end
-
Regula KM, Kirshenbaum LA. Apoptosis of ventricular myocytes: a means to an end. J Mol Cell Cardiol 2005;38:3-13.
-
(2005)
J Mol Cell Cardiol
, vol.38
, pp. 3-13
-
-
Regula, K.M.1
Kirshenbaum, L.A.2
-
9
-
-
23744471140
-
Mitochondria and ischaemia/reperfusion injury
-
Honda HM, Korge P, Weiss JN. Mitochondria and ischaemia/reperfusion injury. Ann NY Acad Sci 2005;1047:248-258.
-
(2005)
Ann NY Acad Sci
, vol.1047
, pp. 248-258
-
-
Honda, H.M.1
Korge, P.2
Weiss, J.N.3
-
10
-
-
0029862706
-
Mitochondrial control of nuclear apoptosis
-
Zamzami N, Susin SA, Marchetti P, Hirsch T, Gomez-Monterrey I, Castedo M et al. Mitochondrial control of nuclear apoptosis. J Exp Med 1996;183:1533-1544.
-
(1996)
J Exp Med
, vol.183
, pp. 1533-1544
-
-
Zamzami, N.1
Susin, S.A.2
Marchetti, P.3
Hirsch, T.4
Gomez-Monterrey, I.5
Castedo, M.6
-
11
-
-
11144293491
-
NHE-1 inhibition improves impaired mitochondrial permeability transition and respiratory function during postinfarction remodelling in the rat
-
Javadov S, Huang C, Kirshenbaum L, Karmazyn M. NHE-1 inhibition improves impaired mitochondrial permeability transition and respiratory function during postinfarction remodelling in the rat. J Mol Cell Cardiol 2005;38:135-143.
-
(2005)
J Mol Cell Cardiol
, vol.38
, pp. 135-143
-
-
Javadov, S.1
Huang, C.2
Kirshenbaum, L.3
Karmazyn, M.4
-
12
-
-
0013672902
-
L-(+)-Lactate
-
Bergmeyer HU ed, New York: Academic Press;
-
Hohorst HJ. L-(+)-Lactate. In: Bergmeyer HU ed. Methods of Enzymatic Analysis. New York: Academic Press; 1963. p215-219.
-
(1963)
Methods of Enzymatic Analysis
, pp. 215-219
-
-
Hohorst, H.J.1
-
13
-
-
0028968606
-
Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion
-
Griffiths EJ, Halestrap AP. Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion. Biochem J 1995;307:93-98.
-
(1995)
Biochem J
, vol.307
, pp. 93-98
-
-
Griffiths, E.J.1
Halestrap, A.P.2
-
14
-
-
77957010982
-
Citrate synthase
-
Srere PA. Citrate synthase. Methods Enzymol 1969;13:3-11.
-
(1969)
Methods Enzymol
, vol.13
, pp. 3-11
-
-
Srere, P.A.1
-
15
-
-
0031013623
-
Oxidative stress, thiol reagents, and membrane potential modulate the mitochondrial permeability transition by affecting nucleotide binding to the adenine nucleotide translocase
-
Halestrap AP, Woodfield KY, Connern CP. Oxidative stress, thiol reagents, and membrane potential modulate the mitochondrial permeability transition by affecting nucleotide binding to the adenine nucleotide translocase. J Biol Chem 1997;272:3346-3354.
-
(1997)
J Biol Chem
, vol.272
, pp. 3346-3354
-
-
Halestrap, A.P.1
Woodfield, K.Y.2
Connern, C.P.3
-
16
-
-
34548495075
-
Induction of renal endonuclease G by cisplatin is reduced in DNase I-deficient mice
-
Yin X, Apostolov EO, Shah SV, Wang X, Bogdanov K, Buzder T et al. Induction of renal endonuclease G by cisplatin is reduced in DNase I-deficient mice. J Am Soc Nephrol 2007;18:2544-2553.
-
(2007)
J Am Soc Nephrol
, vol.18
, pp. 2544-2553
-
-
Yin, X.1
Apostolov, E.O.2
Shah, S.V.3
Wang, X.4
Bogdanov, K.5
Buzder, T.6
-
17
-
-
33749338470
-
NHE-1 inhibition improves cardiac mitochondrial function through regulation of mitochondrial biogenesis during postinfarction remodeling
-
Javadov S, Purdham DM, Zeidan A, Karmazyn M. NHE-1 inhibition improves cardiac mitochondrial function through regulation of mitochondrial biogenesis during postinfarction remodeling. Am J Physiol Heart Circ Physiol 2006;291:H1722-H1730.
-
(2006)
Am J Physiol Heart Circ Physiol
, vol.291
-
-
Javadov, S.1
Purdham, D.M.2
Zeidan, A.3
Karmazyn, M.4
-
24
-
-
0342265145
-
Na(+)/H(+) exchange inhibition with HOE642 improves postischaemic recovery due to attenuation of Ca(2+) overload and prolonged acidosis on reperfusion
-
Stromer H, de Groot MC, Horn M, Faul C, Leupold A, Morgan JP et al. Na(+)/H(+) exchange inhibition with HOE642 improves postischaemic recovery due to attenuation of Ca(2+) overload and prolonged acidosis on reperfusion. Circulation 2000;101:2749-2755.
-
(2000)
Circulation
, vol.101
, pp. 2749-2755
-
-
Stromer, H.1
de Groot, M.C.2
Horn, M.3
Faul, C.4
Leupold, A.5
Morgan, J.P.6
-
25
-
-
1142309745
-
i in rat hearts explain its lack of cardioprotection
-
i in rat hearts explain its lack of cardioprotection. Cardiovasc Res 2004;61:522-529.
-
(2004)
Cardiovasc Res
, vol.61
, pp. 522-529
-
-
Ten Hove, M.1
Van Echteld, C.J.2
-
26
-
-
0032988868
-
Reversal of permeability transition during recovery of hearts from ischaemia and its enhancement by pyruvate
-
Kerr PM, Suleiman MS, Halestrap AP. Reversal of permeability transition during recovery of hearts from ischaemia and its enhancement by pyruvate. Am J Physiol 1999;276:H496-H502.
-
(1999)
Am J Physiol
, vol.276
-
-
Kerr, P.M.1
Suleiman, M.S.2
Halestrap, A.P.3
-
27
-
-
33646771019
-
+ exchanger isoform 1 inhibition is mediated by reduced mitogen-activated protein kinase activation secondary to improved mitochondrial integrity and decreased generation of mitochondrial-derived reactive oxygen species
-
+ exchanger isoform 1 inhibition is mediated by reduced mitogen-activated protein kinase activation secondary to improved mitochondrial integrity and decreased generation of mitochondrial-derived reactive oxygen species. J Pharmacol Exp Ther 2006;317:1036-1043.
-
(2006)
J Pharmacol Exp Ther
, vol.317
, pp. 1036-1043
-
-
Javadov, S.1
Baetz, D.2
Rajapurohitam, V.3
Zeidan, A.4
Kirshenbaum, L.A.5
Karmazyn, M.6
-
29
-
-
0041859202
-
Cariporide preserves mitochondrial proton gradient and delays ATP depletion in cardiomyocytes during ischaemic conditions
-
Ruiz-Meana M, Garcia-Dorado D, Pina P, Inserte J, Agullo L, Soler-Soler J. Cariporide preserves mitochondrial proton gradient and delays ATP depletion in cardiomyocytes during ischaemic conditions. Am J Physiol Heart Circ Physiol 2003;285:H999-H1006.
-
(2003)
Am J Physiol Heart Circ Physiol
, vol.285
-
-
Ruiz-Meana, M.1
Garcia-Dorado, D.2
Pina, P.3
Inserte, J.4
Agullo, L.5
Soler-Soler, J.6
-
32
-
-
0042237031
-
Mitochondrial release of AIF and EndoG requires caspase activation downstream of Bax/Bak-mediated permeabilization
-
Arnoult D, Gaume B, Karbowski M, Sharpe JC, Cecconi F, Youle RJ. Mitochondrial release of AIF and EndoG requires caspase activation downstream of Bax/Bak-mediated permeabilization. EMBO J 2003;22:4385-4399.
-
(2003)
EMBO J
, vol.22
, pp. 4385-4399
-
-
Arnoult, D.1
Gaume, B.2
Karbowski, M.3
Sharpe, J.C.4
Cecconi, F.5
Youle, R.J.6
-
33
-
-
33745250108
-
EndoG is dispensable in embryogenesis and apoptosis
-
David KK, Sasaki M, Yu SW, Dawson TM, Dawson VL. EndoG is dispensable in embryogenesis and apoptosis. Cell Death Differ 2006;13:1147-1155.
-
(2006)
Cell Death Differ
, vol.13
, pp. 1147-1155
-
-
David, K.K.1
Sasaki, M.2
Yu, S.W.3
Dawson, T.M.4
Dawson, V.L.5
-
34
-
-
0035823211
-
Role of intracellular thiol depletion, mitochondrial dysfunction and reactive oxygen species in Salvia miltiorrhizainduced apoptosis in human hepatoma HepG2 cells
-
Liu J, Shen HM, Ong CN. Role of intracellular thiol depletion, mitochondrial dysfunction and reactive oxygen species in Salvia miltiorrhizainduced apoptosis in human hepatoma HepG2 cells. Life Sci. 2001;69:1833-1850.
-
(2001)
Life Sci
, vol.69
, pp. 1833-1850
-
-
Liu, J.1
Shen, H.M.2
Ong, C.N.3
-
35
-
-
0345276715
-
Caspase-dependent apoptosis and -independent poly(ADP-ribose) polymerase cleavage induced by transforming growth factor beta1
-
Yang Y, Zhao S, Song J. Caspase-dependent apoptosis and -independent poly(ADP-ribose) polymerase cleavage induced by transforming growth factor beta1. Int J Biochem Cell Biol 2004;36:223-234.
-
(2004)
Int J Biochem Cell Biol
, vol.36
, pp. 223-234
-
-
Yang, Y.1
Zhao, S.2
Song, J.3
-
36
-
-
34247602459
-
Hypoxia-induced apoptosis and tube breakdown are regulated by p38 MAPK but not by caspase cascade in an in vitro capillary model composed of human endothelial cells
-
Ohta T, Eguchi R, Suzuki A, Miyakaze S, Ayuzawa R, Kaji K. Hypoxia-induced apoptosis and tube breakdown are regulated by p38 MAPK but not by caspase cascade in an in vitro capillary model composed of human endothelial cells. J Cell Physiol 2007;211:673-681.
-
(2007)
J Cell Physiol
, vol.211
, pp. 673-681
-
-
Ohta, T.1
Eguchi, R.2
Suzuki, A.3
Miyakaze, S.4
Ayuzawa, R.5
Kaji, K.6
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