Lysosome mediated Kir2.1 breakdown directly influences inward rectifier current density

Biochemical and Biophysical Research Communications

Volumn 367, Issue 3, 2008, Pages 687-692

  Jansen, John A ^a   de Boer, Teun P ^a   Wolswinkel, Rianne ^a   van Veen, Toon A B ^a   Vos, Marc A ^a   van Rijen, Harold V M ^a   van der Heyden, Marcel A G ^a  

^a UNIVERSITY MEDICAL CENTER UTRECHT   (Netherlands)

Author keywords

Chloroquine; Degradation; Inward rectifier; Kir2.1; Lysosome

Indexed keywords

AMMONIUM CHLORIDE; CADHERIN; CHLOROQUINE; INWARDLY RECTIFYING POTASSIUM CHANNEL; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR2.1; LEUPEPTIN; UNCLASSIFIED DRUG;

ARTICLE; CONTROLLED STUDY; HUMAN; HUMAN CELL; IMMUNODETECTION; LYSOSOME; MEMBRANE STEADY POTENTIAL; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN FUNCTION;

AMMONIUM CHLORIDE; ANIMALS; CELLS, CULTURED; CHLOROQUINE; FLUORESCENT ANTIBODY TECHNIQUE; HUMANS; INTRACELLULAR FLUID; LEUPEPTINS; LYSOSOMES; MICE; PATCH-CLAMP TECHNIQUES; POTASSIUM CHANNELS, INWARDLY RECTIFYING; TIME FACTORS;

EID: 38649085314     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2007.12.168     Document Type: Article

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