Functional role of inward rectifier current in heart probed by Kir2.1 overexpression and dominant-negative suppression

Journal of Clinical Investigation

Volumn 111, Issue 10, 2003, Pages 1529-1536

  Miake, Junichiro ^a,c   Marbán, Eduardo ^a   Nuss, H Bradley ^a,b  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

^c TOTTORI UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BARIUM ION; INWARDLY RECTIFYING POTASSIUM CHANNEL;

ACTION POTENTIAL; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; DOWN REGULATION; ELECTROCARDIOGRAM; GENE DELIVERY SYSTEM; GENE OVEREXPRESSION; GENE TRANSFER; GENETIC TRANSDUCTION; GLYCOGEN STORAGE DISEASE TYPE 4; GUINEA PIG; HEART FAILURE; HEART MUSCLE CELL; HEART RATE; HEART REPOLARIZATION; HYPERPOLARIZATION; IN VIVO STUDY; INWARDLY RECTIFYING CURRENT IK1; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR2.1; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR2.1AAA; MEMBRANE STEADY POTENTIAL; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; QT INTERVAL; SINUS NODE; VIRAL GENE DELIVERY SYSTEM; ADENOVIRUS; ANIMAL; BIOSYNTHESIS; CELL LINE; CELL SEPARATION; DOMINANT GENE; ELECTROCARDIOGRAPHY; FEMALE; GENETICS; HEART; HEART MUSCLE; HUMAN; METABOLISM; MUTATION; PATCH CLAMP; PHYSIOLOGY;

ACTION POTENTIALS; ADENOVIRIDAE; ANIMALS; CELL LINE; CELL SEPARATION; ELECTROCARDIOGRAPHY; FEMALE; GENE TRANSFER TECHNIQUES; GENES, DOMINANT; GUINEA PIGS; HEART; HUMANS; MUTATION; MYOCARDIUM; MYOCYTES, CARDIAC; PATCH-CLAMP TECHNIQUES; PHENOTYPE; POTASSIUM CHANNELS, INWARDLY RECTIFYING; TRANSDUCTION, GENETIC;

EID: 0037975497     PISSN: 00219738     EISSN: None     Source Type: Journal    
DOI: 10.1172/JCI200317959     Document Type: Article

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