Comparative analysis of genetic modifiers in drosophila points to common and distinct mechanisms of pathogenesis among polyglutamine diseases

Human Molecular Genetics

Volumn 17, Issue 3, 2008, Pages 376-390

  Branco, Joana ^a,b   Al ramahi, Ismael ^a   Ukani, Lubna ^a   Pérez, Alma M ^a   Fernandez funez, Pedro ^a,b   Rincón Limas, Diego ^a,b   Botas, Juan ^a,c  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

^b UNIVERSITY OF PORTO   (Portugal)

^c University of Texas Medical Branch School of Medicine   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ATAXIN 1; HUNTINGTIN;

ANIMAL TISSUE; ARTICLE; COMPARATIVE STUDY; CONTROLLED STUDY; DISEASE MODEL; DROSOPHILA; GENE FUNCTION; GENETIC DISORDER; HUNTINGTON CHOREA; NEUROPATHOLOGY; NEUROTOXICITY; NONHUMAN; POLYGLUTAMINE DISEASE; PRIORITY JOURNAL; SPINOCEREBELLAR DEGENERATION;

ANIMALS; ANIMALS, GENETICALLY MODIFIED; DISEASE MODELS, ANIMAL; DROSOPHILA; DROSOPHILA PROTEINS; GENES, DOMINANT; GENES, INSECT; HEREDODEGENERATIVE DISORDERS, NERVOUS SYSTEM; HUMANS; HUNTINGTON DISEASE; NERVE TISSUE PROTEINS; NUCLEAR PROTEINS; PEPTIDES; PHENOTYPE; PHOSPHOLIPID TRANSFER PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT; RECOMBINANT PROTEINS; SPINOCEREBELLAR ATAXIAS;

EID: 38349171781     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/ddm315     Document Type: Article

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