Erratum (DOI:10.1016/j.semnephrol.2007.10.006);Racial and Genetic Factors in IgA Nephropathy

Seminars in Nephrology

Volumn 28, Issue 1, 2008, Pages 48-57

  Hsu, Stephen I Hong ^a  

^a UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

Author keywords

ethnicity; genetics; IgA nephropathy; race

Indexed keywords

DIPEPTIDYL CARBOXYPEPTIDASE;

ARTICLE; CASE CONTROL STUDY; DISEASE COURSE; GENE DELETION; GENE INSERTION; GENE LINKAGE DISEQUILIBRIUM; GENE REPLICATION; GENETIC ASSOCIATION; GENETIC IDENTIFICATION; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENETICS; HAPLOTYPE; HEREDITY; HUMAN; IMMUNOGLOBULIN A NEPHROPATHY; IMMUNOPATHOGENESIS; INCIDENCE; KIDNEY FAILURE; LINKAGE ANALYSIS; MOLECULAR GENETICS; NONHUMAN; PRIORITY JOURNAL; QUANTITATIVE TRAIT LOCUS; RACE DIFFERENCE;

EID: 38349153312     PISSN: 02709295     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.semnephrol.2008.02.002     Document Type: Erratum

References (62)

1. D'Amico G. The commonest glomerulonephritis in the world: IgA nephropathy. QJM 64 (1987) 709-727
2. Julian B.A., Waldo F.B., Rifai A., et al. IgA nephropathy, the most common glomerulonephritis worldwide. A neglected disease in the United States?. Am J Med 84 (1988) 129-132
3. Levy M., and Berger J. Worldwide perspective of IgA nephropathy. Am J Kidney Dis 12 (1988) 340-347
4. Schena F.P. A retrospective analysis of the natural history of primary IgA nephropathy worldwide. Am J Med 89 (1990) 209-215
5. Sinniah R., Law C.H., and Pwee H.S. Glomerular lesions in patients with asymptomatic persistent and orthostatic proteinuria discovered on routine medical examination. Clin Nephrol 7 (1977) 1-14
6. Sinniah R., Pwee H.S., and Lim C.H. Glomerular lesions in asymptomatic microscopic hematuria discovered on routine medical examination. Clin Nephrol 5 (1976) 216-228
7. Sinniah R. Occurrence of mesangial IgA and IgM deposits in a control necropsy population. J Clin Pathol 36 (1983) 276-279
8. Freedman B.I., Soucie J.M., and McClellan W.M. Family history of end-stage renal disease among incident dialysis patients. J Am Soc Nephrol 8 (1997) 1942-1945
9. Feehally J. Ethnicity and renal disease: questions and challenges. Clin Med 3 (2003) 578-582
10. Galla J.H., Kohaut E.C., Alexander R., et al. Racial difference in the prevalence of IgA-associated nephropathies. Lancet 2 (1984) 522
11. Jennette J.C., Wall S.D., and Wilkman A.S. Low incidence of IgA nephropathy in blacks. Kidney Int 28 (1985) 944-950
12. Braden G.L., Mulhern J.G., O'Shea M.H., et al. Changing incidence of glomerular diseases in adults. Am J Kidney Dis 35 (2000) 878-883
13. Dragovic D., Rosenstock J.L., Wahl S.J., et al. Increasing incidence of focal segmental glomerulosclerosis and an examination of demographic patterns. Clin Nephrol 63 (2005) 1-7
14. Korbet S.M., Genchi R.M., Borok R.Z., et al. The racial prevalence of glomerular lesions in nephrotic adults. Am J Kidney Dis 27 (1996) 647-651
15. Nair R., and Walker P.D. Is IgA nephropathy the commonest primary glomerulopathy among young adults in the USA?. Kidney Int 69 (2006) 1455-1458
16. Simon P., Ramee M.P., Boulahrouz R., et al. Epidemiologic data of primary glomerular diseases in western France. Kidney Int 66 (2004) 905-908
17. Stratta P., Segoloni G.P., Canavese C., et al. Incidence of biopsy-proven primary glomerulonephritis in an Italian province. Am J Kidney Dis 27 (1996) 631-639
18. Covic A., Schiller A., Volovat C., et al. Epidemiology of renal disease in Romania: a 10 year review of two regional renal biopsy databases. Nephrol Dial Transplant 21 (2006) 419-424
19. Polenakovic M.H., Grcevska L., and Dzikova S. The incidence of biopsy-proven primary glomerulonephritis in the Republic of Macedonia-long-term follow-up. Nephrol Dial Transplant 18 Suppl 5 (2003) v26-v27
20. Rychlik I., Jancova E., Tesar V., et al. The Czech registry of renal biopsies. Occurrence of renal diseases in the years 1994-2000. Nephrol Dial Transplant 19 (2004) 3040-3049
21. Choi I.J., Jeong H.J., Han D.S., et al. An analysis of 4,514 cases of renal biopsy in Korea. Yonsei Med J 42 (2001) 247-254
22. Koyama A., Igarashi M., and Kobayashi M. Natural history and risk factors for immunoglobulin A nephropathy in Japan. Research Group on Progressive Renal Diseases. Am J Kidney Dis 29 (1997) 526-532
23. Lai F.M., Lai K.N., Chan K.W., et al. Pattern of glomerulonephritis in Hong Kong. Pathology 19 (1987) 247-252
24. Li L.S., and Liu Z.H. Epidemiologic data of renal diseases from a single unit in China: analysis based on 13,519 renal biopsies. Kidney Int 66 (2004) 920-923
25. Woo K.T., Chiang G.S., Pall A., et al. The changing pattern of glomerulonephritis in Singapore over the past two decades. Clin Nephrol 52 (1999) 96-102
26. Frimat L., and Kessler M. Controversies concerning the importance of genetic polymorphism in IgA nephropathy. Nephrol Dial Transplant 17 (2002) 542-545
27. Johnstone D.B., and Holzman L.B. Clinical impact of research on the podocyte slit diaphragm. Nat Clin Pract Nephrol 2 (2006) 271-282
28. Helms C., Cao L., Krueger J.G., et al. A putative RUNX1 binding site variant between SLC9A3R1 and NAT9 is associated with susceptibility to psoriasis. Nat Genet 35 (2003) 349-356
29. Prokunina L., Castillejo-Lopez C., Oberg F., et al. A regulatory polymorphism in PDCD1 is associated with susceptibility to systemic lupus erythematosus in humans. Nat Genet 32 (2002) 666-669
30. Tokuhiro S., Yamada R., Chang X., et al. An intronic SNP in a RUNX1 binding site of SLC22A4, encoding an organic cation transporter, is associated with rheumatoid arthritis. Nat Genet 35 (2003) 341-348
31. Hoogendoorn B., Coleman S.L., Guy C.A., et al. Functional analysis of human promoter polymorphisms. Hum Mol Genet 12 (2003) 2249-2254
32. Lo H.S., Wang Z., Hu Y., et al. Allelic variation in gene expression is common in the human genome. Genome Res 13 (2003) 1855-1862
33. Hsu S.I. The molecular pathogenesis and experimental therapy of IgA nephropathy: recent advances and future directions. Curr Mol Med 1 (2001) 183-196
34. Hsu S.I., Ramirez S.B., Winn M.P., et al. Evidence for genetic factors in the development and progression of IgA nephropathy. Kidney Int 57 (2000) 1818-1835
35. Izzi C., Sanna-Cherchi S., Prati E., et al. Familial aggregation of primary glomerulonephritis in an Italian population isolate: Valtrompia study. Kidney Int 69 (2006) 1033-1040
36. Julian B.A., Quiggins P.A., Thompson J.S., et al. Familial IgA nephropathy. Evidence of an inherited mechanism of disease. N Engl J Med 312 (1985) 202-208
37. Scolari F. Inherited forms of IgA nephropathy. J Nephrol 16 (2003) 317-320
38. Schena F.P., Cerullo G., Rossini M., et al. Increased risk of end-stage renal disease in familial IgA nephropathy. J Am Soc Nephrol 13 (2002) 453-460
39. Izzi C., Ravani P., Torres D., et al. IgA nephropathy: the presence of familial disease does not confer an increased risk for progression. Am J Kidney Dis 47 (2006) 761-769
40. Mittal B.V., Singh A.K., and Rennke H.G. Spectrum of glomerular disease: results of the Brigham database of glomerulonephritis. J Am Soc Nephrol 15 (2004) 553A
41. Gharavi A.G., Yan Y., Scolari F., et al. IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23. Nat Genet 26 (2000) 354-357
42. Tsukaguchi H. A genetic mapping for a familial IgA nephropathy. Nephrology (Carlton) 9 Suppl 2 (2004) A65
43. Schena F.P., Cerullo G., Torres D.D., et al. The IgA nephropathy Biobank. An important starting point for the genetic dissection of a complex trait. BMC Nephrol 6 (2005) 14
44. Bisceglia L., Cerullo G., Forabosco P., et al. Genetic heterogeneity in Italian families with IgA nephropathy: suggestive linkage for two novel IgA nephropathy loci. Am J Hum Genet 79 (2006) 1130-1134
45. Paterson A.D., Liu X.Q., Wang K., et al. Genome-wide linkage scan of a large family with IgA nephropathy localizes a novel susceptibility locus to chromosome 2q36. J Am Soc Nephrol 18 (2007) 2408-2415
46. Suzuki H., Suzuki Y., Yamanaka T., et al. Genome-wide scan in a novel IgA nephropathy model identifies a susceptibility locus on murine chromosome 10, in a region syntenic to human IGAN1 on chromosome 6q22-23. J Am Soc Nephrol 16 (2005) 1289-1299
47. Oida E., Nogaki F., Kobayashi I., et al. Quantitative trait loci (QTL) analysis reveals a close linkage between the hinge region and trimeric IgA dominancy in a high IgA strain (HIGA) of ddY mice. Eur J Immunol 34 (2004) 2200-2208
48. Nogaki F., Oida E., Kamata T., et al. Chromosomal mapping of hyperserum IgA and glomerular IgA deposition in a high IgA (HIGA) strain of DdY mice. Kidney Int 68 (2005) 2517-2525
49. Schneider B., Hanke P., Jagla W., et al. Synergistic interaction of two independent genetic loci causes extreme elevation of serum IgA in mice. Genes Immun 5 (2004) 375-380
50. Schena F.P., D'Altri C., Cerullo G., et al. ACE gene polymorphism and IgA nephropathy: an ethnically homogeneous study and a meta-analysis. Kidney Int 60 (2001) 732-740
51. Rigat B., Hubert C., Alhenc-Gelas F., et al. An insertion/deletion polymorphism in the angiotensin I-converting enzyme gene accounting for half the variance of serum enzyme levels. J Clin Invest 86 (1990) 1343-1346
52. Hunley T.E., Julian B.A., Phillips J.A., et al. Angiotensin converting enzyme gene polymorphism: potential silencer motif and impact on progression in IgA nephropathy. Kidney Int 49 (1999) 571-577
53. Kunz R., Bork J.P., Fritsche L., et al. Association between the angiotensin-converting enzyme-insertion/deletion polymorphism and diabetic nephropathy: a methodologic appraisal and systematic review. J Am Soc Nephrol 9 (1998) 1653-1663
54. Poch E., Gonzalez D., Giner V., et al. Molecular basis of salt sensitivity in human hypertension. Evaluation of renin-angiotensin-aldosterone system gene polymorphisms. Hypertension 38 (2001) 1204-1209
55. Goldstein D.B. Islands of linkage disequilibrium. Nat Genet 29 (2001) 109-111
56. Wahl J.D., and Pritchard J.K. Haplotype blocks and linkage disequilibrium in the human genome. Nat Rev Genet 4 (2003) 587-597
57. Couser W.G. Revisions to instructions to JASN authors regarding articles reporting studies using DNA arrays, DNA polymorphisms and randomized controlled clinical trials. J Am Soc Nephrol 14 (2003) 2686-2687
58. Takei T., Iida A., Nitta K., et al. Association between single-nucleotide polymorphisms in selectin genes and immunoglobulin A nephropathy. Am J Hum Genet 70 (2002) 781-786
59. Takei T., Hiraoka M., Nitta K., et al. Functional impact of IgA nephropathy-associated selectin gene haplotype on leukocyte-endothelial interaction. Immunogenetics 58 (2006) 355-361
60. Li Y.J., Du Y., Li C.X., et al. Family-based association study showing that immunoglobulin A nephropathy is associated with the polymorphisms 2093C and 2180T in the 3' untranslated region of the Megsin gene. J Am Soc Nephrol 15 (2004) 1739-1743
61. Xia Y.F., Huang S., Li X., et al. A family-based association study of megsin A23167G polymorphism with susceptibility and progression of IgA nephropathy in a Chinese population. Clin Nephrol 65 (2006) 153-159
62. Schena F.P., Cerullo G., Torres D.D., et al. Role of interferon-gamma gene polymorphisms in susceptibility to IgA nephropathy: a family-based association study. Eur J Hum Genet 14 (2006) 488-496